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Protein excretion of 150 mg is within the normal range.

Protein excretion of 250 mg is slightly above the normal range, but not enough to indicate proteinuria.

Protein excretion of 350 mg is also above the normal range, but not enough to indicate proteinuria.

No accelerations or decelerations in a 20-minute period indicate a non-reactive test, which may suggest fetal hypoxia or acidosis.

One acceleration of at least 10 beats/min above baseline lasting for at least 10 seconds in a 20-minute period is the criterion for a reactive test for gestational age less than 32 weeks, not 34 weeks.

Variable decelerations with normal variability in a 20-minute period indicate cord compression or fetal head compression, not a reactive test.

Urine output of 50 mL/hr is within the normal range and does not indicate magnesium toxicity.The nurse should monitor the client’s urine output closely and report any decrease below 30 mL/hr.

Serum magnesium level of 6 mg/dL is within the therapeutic range of 4 to 7 mg/dL for clients receiving magnesium sulfate.The nurse should monitor the client’s serum magnesium level regularly and report any increase above 8 mg/dL, which indicates toxicity.

Patellar reflex of 2+ is normal and does not indicate magnesium toxicity.The nurse should assess the client’s deep tendon reflexes frequently and report any decrease or absence of reflexes, which indicates toxicity.

Magnesium sulfate does not lower blood pressure.It may be given along with medications that help reduce blood pressure.

Magnesium sulfate does not induce labor.It may be given to delay delivery for up to 48 hours to allow time for the administration of drugs that speed up the baby’s lung development.

Magnesium sulfate does not reduce edema.Edema is a common symptom of preeclampsia, but it is not a direct cause of complications.

Headache is not a sign of magnesium toxicity.

It may be a symptom of preeclampsia or other conditions, but it does not indicate an overdose of magnesium sulfate.

Nausea is not a sign of magnesium toxicity.

It may be a side effect of magnesium sulfate or a symptom of preeclampsia or other conditions, but it does not indicate an overdose of magnesium sulfate.

Blurred vision is not a sign of magnesium toxicity.

It may be a symptom of preeclampsia or other conditions, but it does not indicate an overdose of magnesium sulfate.

It is the target blood pressure goal for a client with chronic hypertension or gestational hypertension without severe features.

It is the target blood pressure goal for a client with mild pre-eclampsia.

It is too high and can increase the risk of maternal and fetal complications such as stroke, eclampsia, placental abruption, and fetal growth restriction.

Corticosteroids do not reduce inflammation in pre-eclampsia.They are used for other inflammatory conditions such as asthma or arthritis.

Corticosteroids do not prevent infection in pre-eclampsia.They can actually increase the risk of infection by suppressing the immune system.

Corticosteroids do not increase platelet count in pre-eclampsia.They can actually decrease the platelet count by causing thrombocytopenia.

Statement is wrong because this is a normal fetal heart rate.The normal range for fetal heart rate is 120-160 beats per minute.

Statement is wrong because this is a normal frequency for uterine contractions during early labor.

The normal range for uterine contractions is 5-10 minutes apart.

Uterine contractions lasting 60 seconds.

Statement is wrong because this is a normal duration for uterine contractions during active labor.

The normal range for uterine contractions is 45-90 seconds long.

A fetal heart rate of 140 beats per minute is within the normal range of 110 to 160 beats per minute.

Uterine contractions every 10 minutes are not abnormal in a client with severe pre-eclampsia who is receiving magnesium sulfate.Magnesium sulfate is used to prevent seizures and lower blood pressure in pre-eclampsia, but it does not stop labor.

Uterine contractions lasting 60 seconds are not a sign of …

Hydralazine does not prevent cerebral edema.

Cerebral edema is a complication of severe preeclampsia or eclampsia that can cause seizures, headaches, and visual disturbances.Hydralazine may lower the blood pressure and reduce the risk of stroke, but it does not directly affect the brain swelling.

Hydralazine does not reduce uterine contractions.

Uterine contractions are stimulated by oxytocin and prostaglandins, which are not affected by hydralazine.Hydralazine may cause reflex tachycardia, which can increase the cardiac output and uterine blood flow, but it does not alter the uterine muscle activity.

Hydralazine does not promote diuresis.

Diuresis is the increased production of urine by the kidneys.

Hydralazine may cause fluid retention and edema by activating the renin-angiotensin-aldosterone system, which increases sodium and water reabsorption.Hydralazine may also cause a decrease in renal perfusion and glomerular filtration rate, which can impair the kidney function and urine output.

This is wrong because inserting an oral airway during a seizure can cause injury to the client’s mouth or teeth, and it can also stimulate the gag reflex and increase the risk of vomiting and aspiration.

This is wrong because administering oxygen via face mask during a seizure can be difficult and ineffective, as the client may not be able to breathe through the mask or may dislodge it with their movements.Oxygen can be given after the seizure has stopped, if needed.

This is wrong because documenting the duration of the seizure is not a priority action during a seizure.The nurse should first ensure the client’s safety and airway patency, and then document the seizure characteristics after it has ended.

Corticosteroids do not lower blood pressure.Antihypertensive drugs such as hydralazine or labetalol are used to treat hypertension in eclampsia.

Corticosteroids do not prevent seizures.Magnesium sulfate is the drug of choice for seizure prophylaxis and treatment in eclampsia.

Corticosteroids do not reduce inflammation in the body.They may have anti-inflammatory effects in some conditions, but their main purpose in eclampsia is to enhance fetal lung development.

Monitoring fetal heart rate and uterine activity continuouslyis not a priority intervention for a client with eclampsia.The priority is to prevent seizures and control blood pressure.

Administering oxytocin to augment laboris contraindicated in a client with eclampsia.Oxytocin can increase blood pressure and cause uterine hyperstimulation, which can worsen the condition and endanger the mother and the fetus.

Encouraging oral fluids and a high-protein dietis not appropriate for a client with eclampsia.

The client should be kept NPO to prevent aspiration in case of a seizure.A high-protein diet can increase the risk of renal failure and hepatic dysfunction.

Assessing for signs of placental abruptionis not a specific intervention for a client with eclampsia.Placental abruption can occur as a complication of eclampsia, but it is not the main focus of care.

Elevated serum creatinine indicates kidney dysfunction, which is not specific for hemolysis.

Elevated serum alkaline phosphatase (ALP) indicates liver damage, which is another component of HELLP syndrome, but not specific for hemolysis.

Elevated serum uric acid indicates increased purine metabolism, which can be associated with preeclampsia and HELLP syndrome, but not specific for hemolysis.