The emergency department nurse evaluates that fluid resuscitation for a 70 kg patient in shock is effective by finding which one of the following?
The patient's mean arterial pressure (MAP) is 50 mm Hg.
The patient's GCS score is 9.
The patient's urine output has been 35 ml over the last hour.
The patient's hemoglobin is within normal limits.
The Correct Answer is C
Urine output is an essential indicator of renal perfusion and overall fluid status. In a patient in shock, maintaining an adequate urine output is a crucial goal of fluid resuscitation. A urine output of 0.5 to 1 mL/kg/hour is generally considered adequate in adults. The given value of 35 ml over the last hour suggests that the patient is producing urine, which indicates that fluid resuscitation is effective in restoring perfusion to the kidneys.
A. The patient's mean arterial pressure (MAP) is 50 mm Hg in (option A) is incorrect because While mean arterial pressure is an important hemodynamic parameter, a single value alone may not provide a comprehensive assessment of the patient's response to fluid resuscitation.
B. The patient's GCS score is 9 in (option B) is incorrect because The Glasgow Coma Scale (GCS) assesses the level of consciousness and neurological function but does not directly reflect fluid resuscitation effectiveness.
D. The patient's hemoglobin is within normal limits: (option D) is incorrect because Haemoglobin levels are important for assessing oxygen-carrying capacity but do not directly indicate the effectiveness of fluid resuscitation.
Therefore, the nurse can evaluate that fluid resuscitation for a 70 kg patient in shock is effective by observing a urine output of 35 ml over the last hour.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Assessing tissue perfusion is crucial in evaluating the adequacy of oxygen and nutrient delivery to the body's tissues. While multiple factors contribute to tissue perfusion, the options provided in choice B are key indicators:
Level of consciousness: Altered mental status or changes in the patient's level of consciousness can be a sign of impaired cerebral perfusion, which reflects overall tissue perfusion.
Urine output: Monitoring urine output provides information about renal perfusion and kidney function. Decreased urine output can be indicative of inadequate tissue perfusion.
Lactate level: Lactate is a by-product of anaerobic metabolism that accumulates when there is insufficient oxygen delivery to tissues. Elevated lactate levels indicate tissue hypoperfusion and cellular oxygen debt.
A. Pupil response, pulse pressure, and urine output in (option A) are incorrect because While pupil response and pulse pressure may provide some information about perfusion, they do not encompass a comprehensive assessment of tissue perfusion. Additionally, assessing urine output is important, but it alone may not provide a complete picture of tissue perfusion status.
C. Blood pressure, pulse, and respirations in (option C) are incorrect because Blood pressure, pulse, and respirations are important vital signs to monitor, but they do not solely indicate tissue perfusion. Hypotension, for example, can be a late sign of inadequate tissue perfusion.
D. Breath sounds, heart rate, and pupil response in (option D) are incorrect because: Although breath sounds and heart rate can be affected by changes in tissue perfusion, they are not specific or comprehensive indicators of tissue perfusion status. Pupil response alone does not provide a complete assessment of tissue perfusion.
Therefore, the most accurate assessment of tissue perfusion in a patient in shock involves evaluating the level of consciousness, urine output, and lactate levels.
Correct Answer is D
Explanation
In the compensatory stage of shock, the body initiates various mechanisms to maintain perfusion to vital organs and restore homeostasis. Activation of the renin-angiotensin system is one of the compensatory responses. The decreased blood flow and oxygen delivery to the kidneys stimulate the release of renin from the kidneys. Renin acts on angiotensinogen, converting it into angiotensin I, which is further converted to angiotensin II by the action of angiotensin-converting enzyme (ACE). Angiotensin II is a potent vasoconstrictor and also stimulates the release of aldosterone, leading to sodium and water retention. These mechanisms aim to increase blood pressure and cardiac output and restore fluid balance.
A. The initial stage of shock in (option A) is incorrect because it is characterized by inadequate tissue perfusion and the activation of various compensatory mechanisms, including the release of stress hormones. However, the renin-angiotensin system is not specifically mentioned as activated in this stage.
B. The progressive stage of shock in (option B) is incorrect because it occurs when compensatory mechanisms fail to maintain adequate perfusion, leading to worsening hypoperfusion and organ dysfunction. The renin-angiotensin system continues to be activated during this stage, but it is primarily associated with the compensatory stage.
C. The refractory stage of shock in (option C) is incorrect because it is the stage of severe and prolonged hypoperfusion, where organ failure becomes irreversible. The renin-angiotensin system may still be activated, but it is not the primary focus of this stage.
Therefore, the activation of the renin-angiotensin system occurs during the compensatory stage of shock.
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