Which of the following is a common cause of cardiogenic shock?
Anaphylaxis
Hypovolemia
Pulmonary embolism
Myocardial infarction
The Correct Answer is D
D. Myocardial infarction (heart attack) is a common cause of cardiogenic shock. In myocardial infarction, part of the heart muscle becomes ischemic or necrotic due to occlusion of a coronary artery. This leads to impaired cardiac function and reduced cardiac output, resulting in cardiogenic shock. Prompt recognition and treatment of myocardial infarction are crucial to prevent or manage cardiogenic shock.
A. Anaphylaxis is a severe allergic reaction that can lead to systemic vasodilation and distributive shock, but it is not a common cause of cardiogenic shock. In anaphylaxis, the primary mechanism of shock is typically related to widespread vasodilation and increased vascular permeability rather than impaired cardiac function.
B. Hypovolemic shock occurs due to a decrease in intravascular volume, leading to inadequate tissue perfusion. It is not a common cause of cardiogenic shock, as the underlying mechanism is different. In hypovolemic shock, the primary issue is the loss of circulating blood volume, whereas cardiogenic shock involves impaired cardiac function.
C. Pulmonary embolism can lead to acute right heart strain or failure, which may result in hemodynamic instability and shock. However, pulmonary embolism typically causes obstructive shock rather than cardiogenic shock. Obstructive shock occurs when blood flow is obstructed, such as by a pulmonary embolism, leading to reduced cardiac output.
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Related Questions
Correct Answer is B
Explanation
B. Sodium nitroprusside is a potent vasodilator used to reduce systemic vascular resistance and afterload, thereby improving cardiac output and tissue perfusion in cardiogenic shock. In this scenario, where the patient has cool and clammy skin with high SVR, indicating peripheral vasoconstriction,
increasing the rate of sodium nitroprusside infusion can help vasodilate peripheral vessels, reduce afterload, and improve tissue perfusion
A. Dopamine is a medication commonly used in the management of cardiogenic shock to increase cardiac output and systemic blood pressure. However, in this scenario where the patient is cool and clammy with high SVR, indicating vasoconstriction and potential peripheral hypoperfusion, increasing the rate of dopamine infusion may further increase systemic vascular resistance and exacerbate peripheral vasoconstriction. This can worsen tissue perfusion and exacerbate the patient's condition.
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C. Nitroglycerin is another vasodilator commonly used in the management of cardiogenic shock to reduce preload and afterload, thereby improving cardiac output and tissue perfusion. However, decreasing the rate of nitroglycerin infusion may further exacerbate vasoconstriction and increase SVR, worsening tissue perfusion in this scenario. Therefore, decreasing the rate of nitroglycerin infusion is not indicated.
D. Intravenous fluids such as 5% dextrose in normal saline are typically administered to maintain adequate intravascular volume and perfusion pressure in shock states. However, decreasing the rate of intravenous fluid infusion may further decrease intravascular volume and preload, potentially exacerbating hypoperfusion and worsening the patient's condition. Therefore, decreasing the rate of intravenous fluid infusion is not indicated in this scenario.
Correct Answer is C
Explanation
C. Hypovolemia, or low blood volume, can lead to decreased venous return to the heart and reduced filling pressures. Consequently, CVP may decrease in hypovolemic states. Low CVP may indicate inadequate preload and reduced cardiac output, which are characteristic of hypovolemia.
A. Left ventricular failure typically results in elevated filling pressures rather than low CVP. In left ventricular failure, blood backs up into the pulmonary circulation, leading to increased pulmonary venous pressure and potentially elevated pulmonary capillary wedge pressure (PCWP), which is a surrogate marker for left atrial pressure. This elevated pressure is reflected in the CVP as well, resulting in increased CVP rather than low CVP.
B. Fluid overload typically results in elevated filling pressures and increased CVP rather than low CVP. Excess fluid volume increases venous return to the heart, leading to increased pressure within the central veins and elevated CVP.
D. Intracardiac shunts may cause alterations in cardiac pressures, but they typically do not result in consistently low CVP. Depending on the type and severity of the shunt, the direction and magnitude of pressure changes may vary. However, in the absence of other pathophysiological factors, intracardiac shunts are less likely to cause consistently low CVP.
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