When admitting a patient with possible respiratory failure and a high PaCO2, which assessment information should be immediately reported to the health care provider?
The patient's blood pressure is 164/98.
The patient appears somnolent.
The patient's oxygen saturation is 90%.
The patient reports feeling weak.
The Correct Answer is B
B Somnolence, or excessive sleepiness, can indicate respiratory depression, hypercapnia (elevated PaCO2), or impending respiratory failure. Somnolence in this context is concerning as it may suggest worsening respiratory status and impending respiratory compromise. Therefore, it should be immediately reported to the healthcare provider for further evaluation and intervention.
A blood pressure of 164/98 mmHg is elevated but may not necessarily require immediate intervention, especially if the patient is not exhibiting signs of acute hypertensive crisis or end-organ damage.
However, it should be closely monitored and managed as appropriate. While hypertension may contribute to respiratory distress, it may not be the most critical finding to report immediately in a patient with possible respiratory failure and a high PaCO2.
C An oxygen saturation of 90% indicates hypoxemia, which can exacerbate respiratory failure and contribute to respiratory distress. While hypoxemia requires prompt intervention to improve oxygenation, it may not be the most critical finding to report immediately if the patient is not showing signs of severe respiratory distress or impending respiratory failure. However, it should be closely monitored and managed to prevent further deterioration.
D Weakness is a nonspecific symptom and may be related to various underlying causes, including respiratory failure, electrolyte imbalances, or systemic illness. While weakness warrants further assessment and management, it may not be the most urgent finding to report immediately in a patient with possible respiratory failure and a high PaCO2 unless it is accompanied by other concerning symptoms or signs of impending respiratory compromise.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
C. Hypovolemia, or low blood volume, can lead to decreased venous return to the heart and reduced filling pressures. Consequently, CVP may decrease in hypovolemic states. Low CVP may indicate inadequate preload and reduced cardiac output, which are characteristic of hypovolemia.
A. Left ventricular failure typically results in elevated filling pressures rather than low CVP. In left ventricular failure, blood backs up into the pulmonary circulation, leading to increased pulmonary venous pressure and potentially elevated pulmonary capillary wedge pressure (PCWP), which is a surrogate marker for left atrial pressure. This elevated pressure is reflected in the CVP as well, resulting in increased CVP rather than low CVP.
B. Fluid overload typically results in elevated filling pressures and increased CVP rather than low CVP. Excess fluid volume increases venous return to the heart, leading to increased pressure within the central veins and elevated CVP.
D. Intracardiac shunts may cause alterations in cardiac pressures, but they typically do not result in consistently low CVP. Depending on the type and severity of the shunt, the direction and magnitude of pressure changes may vary. However, in the absence of other pathophysiological factors, intracardiac shunts are less likely to cause consistently low CVP.
Correct Answer is B
Explanation
B. Tachycardia is a compensatory mechanism aimed at increasing cardiac output and maintaining tissue perfusion. In response to decreased perfusion, the body increases heart rate to improve circulation and oxygen delivery to tissues.
A. Hypokalemia, or low potassium levels, is not typically associated with the compensatory stage of shock. Instead, electrolyte imbalances may develop later in the progression of shock due to tissue hypoperfusion and metabolic disturbances.
C. Mottled skin, characterized by patchy discoloration due to uneven blood flow, is more commonly seen in the progressive or irreversible stages of shock. It indicates significant tissue hypoperfusion and impaired microcirculation, rather than the compensatory stage
D. This blood pressure reading is within the normal range and may be maintained during the compensatory stage of shock. Initially, blood pressure may be maintained or only slightly decreased due to compensatory mechanisms such as vasoconstriction. However, blood pressure can decrease further as shock progresses beyond the compensatory stage.
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