A patient develops increasing dyspnea and hypoxemia 2 days after heart surgery. What procedure should the nurse anticipate assisting with to determine whether the patient has acute respiratory distress syndrome (ARDS) or pulmonary edema caused by heart failure?

B. This is a reasonable action given the client's respiratory rate and potential hypoxemia resulting from the sucking chest wound. Providing supplemental oxygen can help improve oxygenation and support the client's respiratory effort. However, it doesn't directly address the underlying cause of the shock.

A. In this scenario, the client's vital signs indicate signs of shock, which could be due to significant blood loss from the sucking chest wound. While inserting a central line may be necessary for administering fluids and medications rapidly, it's not the immediate priority in this situation. Stabilizing the client's condition takes precedence.

C. Elevating the foot of the bed to a 90° angle is not appropriate in this situation. This position can further decrease venous return to the heart, potentially exacerbating the client's hypotension and shock. It's crucial to maintain a neutral or slightly elevated position to optimize venous return.

D. While it's important to assess the wound and monitor for any changes, removing the dressing on a sucking chest wound without appropriate precautions can worsen the client's condition. The dressing helps to maintain a seal over the wound, preventing further air from entering the pleural space and worsening the tension pneumothorax. Removing the dressing should be done cautiously and preferably by a healthcare provider trained in managing chest trauma.

D. Myocardial infarction (heart attack) is a common cause of cardiogenic shock. In myocardial infarction, part of the heart muscle becomes ischemic or necrotic due to occlusion of a coronary artery. This leads to impaired cardiac function and reduced cardiac output, resulting in cardiogenic shock. Prompt recognition and treatment of myocardial infarction are crucial to prevent or manage cardiogenic shock.
A. Anaphylaxis is a severe allergic reaction that can lead to systemic vasodilation and distributive shock, but it is not a common cause of cardiogenic shock. In anaphylaxis, the primary mechanism of shock is typically related to widespread vasodilation and increased vascular permeability rather than impaired cardiac function.
B. Hypovolemic shock occurs due to a decrease in intravascular volume, leading to inadequate tissue perfusion. It is not a common cause of cardiogenic shock, as the underlying mechanism is different. In hypovolemic shock, the primary issue is the loss of circulating blood volume, whereas cardiogenic shock involves impaired cardiac function.
C. Pulmonary embolism can lead to acute right heart strain or failure, which may result in hemodynamic instability and shock. However, pulmonary embolism typically causes obstructive shock rather than cardiogenic shock. Obstructive shock occurs when blood flow is obstructed, such as by a pulmonary embolism, leading to reduced cardiac output.