An ulcer caused by H. pylori can be successfully treated with which classification of drugs?

A) This is normal.
Guaifenesin is an expectorant medication commonly used to help loosen and thin mucus in the airways, making it easier to cough up. Coughing up phlegm after taking guaifenesin is an expected response and indicates that the medication is working as intended. Guaifenesin helps to clear the airways of excess mucus, which can be particularly beneficial for individuals with respiratory conditions such as bronchitis or chest congestion.

B) This requires further investigation.
Coughing up phlegm after taking guaifenesin is a common and expected response and does not typically indicate a need for further investigation unless there are other concerning symptoms present. It is important for the nurse to provide reassurance and education about the expected effects of the medication.

C) This is indicative of an allergic reaction.
Coughing up phlegm after taking guaifenesin is not indicative of an allergic reaction. Allergic reactions to guaifenesin are rare but may include symptoms such as rash, itching, swelling, dizziness, or difficulty breathing. If the client were experiencing these symptoms, it would be important to assess further for a possible allergic reaction and intervene accordingly.

D) This means there is probably more pathology present.
Coughing up phlegm after taking guaifenesin does not necessarily indicate the presence of more pathology. It simply indicates that the medication is helping to loosen and mobilize mucus in the airways, which is a normal response to expectorant therapy. If the client has concerns about their symptoms, further assessment by a healthcare provider may be warranted, but coughing up phlegm alone is not necessarily indicative of worsening pathology.

A. Family history: Having a family history of peptic ulcers increases the risk of developing the condition, suggesting a genetic predisposition.

B. Blood type A: There is no direct association between blood type A and peptic ulcer disease.

C. Acetaminophen (Tylenol) intake for pain: Acetaminophen is generally considered safe for pain relief and is not a significant risk factor for peptic ulcer disease. However, nonsteroidal anti-inflammatory drugs (NSAIDs) such as aspirin, ibuprofen, and naproxen are known to increase the risk of peptic ulcers.

D. Smoking tobacco: Smoking tobacco is a significant risk factor for peptic ulcer disease. Tobacco use increases stomach acid production, weakens the protective lining of the stomach and duodenum, and impairs the healing of ulcers.

E. Drinking caffeine: While caffeine consumption alone may not directly cause peptic ulcers, excessive intake of caffeinated beverages such as coffee, tea, and soda can aggravate existing ulcers by stimulating stomach acid production and increasing gastric acidity.