A nurse works with a patient to establish goals/outcomes. The nurse believes that one outcome suggested by the patient is not in the patient’s best interest. What is the nurse’s best action?

Choice A reason: Claiming all mental illnesses can be cured oversimplifies disorders like schizophrenia, which often involve chronic dopamine dysregulation in the prefrontal cortex and limbic system, requiring lifelong management. This approach ignores genetic predispositions and neurobiological complexities, potentially fostering false hope and stigma by implying recovery is universal, disregarding evidence of persistent symptoms in many cases.

Choice B reason: Highlighting creativity and freedom romanticizes mental illnesses, ignoring their debilitating effects. Disorders like bipolar disorder may show creativity in manic phases, but hypomania often impairs judgment and stability. Neuroimaging shows altered amygdala activity in such conditions, causing emotional dysregulation. This portrayal minimizes suffering and misrepresents the neurobiological basis, perpetuating misunderstanding and stigma.

Choice C reason: Correcting misperceptions educates about the neurobiological underpinnings of mental illnesses, such as serotonin imbalances in depression or GABA deficits in anxiety. By addressing myths, nurses promote understanding that these are medical conditions involving brain dysfunction, not personal failings. This fosters empathy, reduces stigma, and aligns with evidence-based approaches to mental health advocacy.

Choice D reason: Attributing most mental illnesses to substance use disorders is inaccurate. While substances can exacerbate symptoms, primary disorders like major depression involve genetic factors and altered neurotransmitter activity, such as reduced serotonin uptake. This oversimplification ignores distinct etiologies, risks misdiagnosis, and perpetuates stigma by blaming patients for their conditions, contrary to scientific evidence.

Choice A reason: Buspirone enhances serotonin activity, taking weeks to reduce anxiety. Panic attacks, driven by acute norepinephrine surges in the amygdala, require rapid intervention. Buspirone’s delayed onset makes it ineffective for acute symptom relief, unlike fast-acting options targeting immediate neurochemical imbalances.

Choice B reason: Venlafaxine, an SNRI, increases serotonin and norepinephrine over weeks, unsuitable for acute panic attacks. Panic involves rapid sympathetic activation, requiring immediate GABA enhancement or similar fast-acting mechanisms, not gradual reuptake inhibition, making venlafaxine incorrect for rapid relief.

Choice C reason: Imipramine, a tricyclic, modulates serotonin and norepinephrine but takes weeks to act. Acute panic, driven by locus coeruleus norepinephrine spikes, needs immediate relief. Imipramine’s slow onset and side effects make it inappropriate for rapid intervention in acute anxiety episodes.

Choice D reason: Alprazolam, a benzodiazepine, enhances GABA-A receptor activity, rapidly inhibiting excessive neural firing in the amygdala during panic attacks. This provides quick relief from acute anxiety symptoms, like tachycardia, within minutes, making it the correct choice for immediate neurobiological stabilization in panic episodes.