Which of the following speech patterns is an example of tangential thinking?
Involuntary, excessive continuation or repetition of a single response or idea
Has a lack of a logical relationship between thoughts, making the speech vague, diffuse, and unfocused
Overproductive speech which rapidly moves from topic to topic with a tenuous logical link between topics
Provides a lot of unnecessary detail, never returns to the central point, and never answers the question
The Correct Answer is D
Choice A reason: Involuntary repetition, or perseveration, involves repeating a single idea, often due to frontal lobe dysfunction in disorders like schizophrenia. Unlike tangential thinking, it fixates on one thought without divergence, making it distinct and incorrect for describing the diffuse, off-point speech of tangentially.
Choice B reason: Lacking logical relationships describes loose associations, not tangential thinking. Loose associations, seen in schizophrenia, reflect disorganized thoughts due to dopamine dysregulation, jumping illogically between ideas. Tangentiality diverges with excessive detail, staying somewhat related but off-point, making this option incorrect.
Choice C reason: Overproductive speech with tenuous links describes flight of ideas, common in mania with elevated dopamine. Unlike tangentiality, it involves rapid topic shifts with loose connections, not excessive detail missing the point. This distinction makes it an incorrect choice for tangential thinking.
Choice D reason: Tangential thinking involves excessive, irrelevant details, failing to return to the original question, often seen in schizophrenia or mania. This reflects disrupted executive function in the prefrontal cortex, impairing focus. The description matches this pattern, making it the correct choice for tangential speech.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Abruptly stopping fluoxetine, an SSRI, disrupts serotonin levels, causing discontinuation syndrome with symptoms like dizziness and irritability due to rapid neurotransmitter imbalance in the brain. Gradual tapering stabilizes serotonin, preventing withdrawal, making this critical teaching for safe medication management in anxiety treatment.
Choice B reason: Constipation is not a common side effect of fluoxetine, which primarily causes nausea or diarrhea via serotonin modulation. Anticholinergic drugs, not SSRIs, typically cause constipation. This teaching is inaccurate, as fluoxetine’s side effect profile does not emphasize gastrointestinal slowing, making it incorrect.
Choice C reason: Fluoxetine takes 4-8 weeks, not months, to reach efficacy by increasing serotonin in the prefrontal cortex and amygdala. Overstating the timeline discourages adherence, as patients expect faster relief from anxiety symptoms, making this teaching point scientifically inaccurate and misleading.
Choice D reason: Fluoxetine is not addictive, as it lacks the reinforcing GABA effects of benzodiazepines. It modulates serotonin for anxiety without dependence risk. This teaching is incorrect, as it misrepresents fluoxetine’s pharmacological profile, potentially causing unnecessary fear about its safe use in treatment.
Correct Answer is A
Explanation
Choice A reason: Blocking norepinephrine at alpha-1 receptors inhibits vasoconstriction, reducing vascular tone. This disrupts baroreceptor-mediated blood pressure regulation, causing orthostatic hypotension when standing. The autonomic nervous system fails to compensate for positional changes, leading to dizziness and fainting, a common side effect of alpha-1 blockers like prazosin.
Choice B reason: Increased psychotic symptoms are linked to dopamine dysregulation, not alpha-1 receptor blockade. Norepinephrine blockade affects autonomic functions, not psychosis, which involves mesolimbic dopamine hyperactivity. This side effect is unrelated to alpha-1 receptors, making this option scientifically inaccurate for the described mechanism.
Choice C reason: Appetite disturbance is typically associated with serotonin or histamine receptor effects, not alpha-1 norepinephrine blockade. Norepinephrine at alpha-1 receptors regulates vascular tone, not appetite control, which involves hypothalamic signaling. This side effect is not a direct consequence of alpha-1 blockade, rendering this option incorrect.
Choice D reason: Hypertensive crisis results from excessive norepinephrine activity, often due to monoamine oxidase inhibitors, not alpha-1 receptor blockade. Blocking alpha-1 receptors causes vasodilation, lowering blood pressure, not raising it. This makes hypertensive crisis an unlikely side effect, contrary to the pharmacological mechanism of alpha-1 blockers.
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