A nurse is planning care for a client who has increased intracranial pressure. The nurse should understand that enteral nutrition should begin within 24 to 48 hr to help prevent which of the following complications?

A. The aortic and mitral valves are the most commonly replaced valves: This statement is accurate. The aortic and mitral valves are indeed the most commonly replaced valves in cardiac valve replacement surgeries. Aortic valve replacement is commonly performed for conditions such as aortic stenosis or regurgitation, while mitral valve replacement is often indicated for mitral regurgitation or stenosis.

B. Mitral valve insufficiency occurs during the diastolic phase of the cardiac cycle: This statement is incorrect. Mitral valve insufficiency, also known as mitral regurgitation, occurs during the systolic phase of the cardiac cycle. It involves the backflow of blood from the left ventricle into the left atrium during ventricular systole, leading to volume overload of the left atrium and potentially causing symptoms such as dyspnea and fatigue.

C. Inadequate closure of the tricuspid valve causes overload in the left ventricle: This statement is incorrect. Inadequate closure of the tricuspid valve leads to regurgitation of blood from the right ventricle back into the right atrium during ventricular systole. This condition, known as tricuspid regurgitation, causes volume overload in the right atrium and ventricle, rather than overload in the left ventricle.

D. Aortic stenosis increases right ventricular systolic pressure and decreases afterload: This statement is partially correct. Aortic stenosis indeed increases left ventricular systolic pressure and decreases afterload, not right ventricular pressure. The increased afterload on the left ventricle can lead to left ventricular hypertrophy and eventually heart failure. However, aortic stenosis does not directly affect right ventricular systolic pressure.

A. Diabetes mellitus: An inverse association between diabetes mellitus (DM) and abdominal aortic aneurysm (AAA) risk hasbeen reported. Apart from a lower AAA prevalence among patients with vsthose without DM, there isdata showing that DM may exert a protective role on aneurysmal growth in patients with small AAAs, thus decreasing the risk of rupture. As atherosclerosis has almost the same risk factors as aneurysms, the decreased AAA prevalence in patients with DM may indicate that atherosclerosis is an associated feature and not a cause of the aneurysms.

B. Total cholesterol 170 mg/dL (less than 200 mg/dL): While elevated total cholesterol is a risk factor for cardiovascular disease, it is not specifically listed as a risk factor for abdominal aortic aneurysm (AAA). However, dyslipidemia, including elevated total cholesterol levels, can contribute to the development of atherosclerosis, which is a risk factor for AAA.

C. HDL cholesterol 65 mg/dL (male greater than 45 mg/dL; female greater than 55 mg/dL): High-density lipoprotein (HDL) cholesterol levels greater than 65 mg/dL are not listed as a risk factor for abdominal aortic aneurysm (AAA). However, low levels of HDL cholesterol are associated with an increased risk of cardiovascular disease, which may indirectly contribute to the development of AAA through the promotion of atherosclerosis.

D. Smoking cigarettes: Smoking cigarettes is a significant modifiable risk factor for abdominal aortic aneurysm (AAA). Smoking damages the walls of blood vessels, promotes inflammation, and accelerates the development of atherosclerosis, increasing the risk of AAA formation and rupture.

E. Family history of aneurysm: A family history of aneurysm, particularly abdominal aortic aneurysm (AAA), is a risk factor for developing AAA. Genetic factors can predispose individuals to the development of aneurysms, and a positive family history increases the likelihood of AAA occurrence.