A nurse is planning care for a client who has increased intracranial pressure. The nurse should understand that enteral nutrition should begin within 24 to 48 hr to help prevent which of the following complications?

A. Completing hourly endotracheal suctioning: Hourly endotracheal suctioning is not typically indicated for a client with increased intracranial pressure (ICP). Frequent suctioning can lead to increased intrathoracic pressure and potentially compromise venous return, which may further elevate ICP. Suctioning should be performed as needed to maintain airway patency while minimizing the risk of increasing ICP.

B. Ensuring proper ventriculostomy transducer levels: Ensuring proper ventriculostomy transducer levels is important for accurate measurement of intracranial pressure (ICP) but may not directly alleviate elevated ICP. Monitoring ICP through ventriculostomy allows for timely detection of changes in ICP, which can guide interventions to manage elevated pressure levels. However, it is not a direct intervention to reduce ICP.

C. Monitoring volume status: Monitoring volume status is important in managing a client with increased intracranial pressure (ICP) as both hypovolemia and hypervolemia can impact ICP. However, monitoring volume status alone does not directly address elevated ICP. Interventions to optimize volume status, such as fluid administration or diuresis, may be implemented based on assessment findings, but they should be done cautiously to avoid exacerbating cerebral edema or altering cerebral perfusion.

D. Elevating the head of the bed 15°: Elevating the head of the bed 15° (or higher) is a crucial intervention for managing a client with increased intracranial pressure (ICP). This position helps promote venous drainage from the brain, reducing venous congestion and intracranial pressure. Elevating the head of the bed also helps prevent cerebrospinal fluid (CSF) from pooling in the brain, which can further increase ICP. Placing the client in a semi-upright position is a standard practice in managing ICP and is recommended in various clinical guidelines.

A. The heart's electrical transmission through the atrioventricular (AV) node is unusually slow: This statement does not align with the manifestations of dizziness and palpitations in atrial fibrillation. A slow conduction through the AV node would typically manifest as bradycardia or heart block rather than rapid and irregular palpitations.

B. The heart's electrical signals are rapid, chaotic, and irregular: This is the correct answer. Atrial fibrillation is characterized by rapid, chaotic, and irregular electrical signals in the atria, leading to ineffective atrial contractions and an irregular ventricular response. The irregularity and rapidity of the heart rate can lead to symptoms such as palpitations and dizziness.

C. An early electrical signal occurs before the expected sinoatrial (SA) node signal: This statement does not accurately describe the mechanism of atrial fibrillation. Atrial fibrillation involves disorganized electrical activity in the atria, rather than a single early electrical signal.

D. The SA node sends an electrical signal greater than 100/min: While a heart rate greater than 100/min (tachycardia) is commonly associated with atrial fibrillation, this statement does not fully explain the manifestations of dizziness and palpitations. These symptoms are more directly related to the irregularity and chaotic nature of the heart's electrical signals in atrial fibrillation.