A 78-kg patient in septic shock has a pulse rate of 120 beats/min with low central venous pressure and pulmonary artery wedge pressure. After initial fluid volume resuscitation, the patient's urine output has been 30 mL/hr for the past 3 hours. Which order by the health care provider should the nurse question?
Give hydrocortisone (Solu-Cortef) 100 mg IV.
Administer furosemide (Lasix) 40 mg IV.
Increase normal saline infusion to 250 ml/hr.
Use norepinephrine to keep systolic BP above 90 mm Hg.
The Correct Answer is B
B. Administering furosemide, a loop diuretic, to a patient in septic shock with low urine output could exacerbate hypovolemia and worsen organ perfusion. It could further reduce preload, exacerbating hypotension, and impairing cardiac output. Loop diuretics are typically contraindicated in hypovolemic shock.
A. Corticosteroids such as hydrocortisone can help improve vascular tone and responsiveness to vasopressors. Given the patient's clinical presentation and lack of response to initial fluid resuscitation, administering hydrocortisone is appropriate.
C. Increasing the fluid infusion rate can help restore intravascular volume and improve organ perfusion. However, caution should be exercised to avoid fluid overload and pulmonary edema.
D .Norepinephrine is a vasopressor commonly used to increase systemic vascular resistance and improve blood pressure in septic shock. It helps maintain perfusion pressure to vital organs, thereby supporting organ function.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["A","B","C","E"]
Explanation
A. Chronic hypertension is a significant risk factor for the development of various cardiac diseases, including coronary artery disease, left ventricular hypertrophy, heart failure, and arrhythmias. The increased workload on the heart caused by hypertension can lead to myocardial remodeling, impaired cardiac function, and ultimately heart disease.
B. Chronic hypertension can cause damage to the kidneys over time, leading to renal insufficiency or chronic kidney disease. The elevated blood pressure damages the delicate blood vessels in the kidneys, impairing their ability to filter waste products and regulate fluid and electrolyte balance. Persistent hypertension can ultimately lead to kidney dysfunction and renal failure.
C. Hypertension is a major risk factor for the development of stroke, particularly ischemic stroke (caused by a blockage in a blood vessel supplying the brain) and hemorrhagic stroke (caused by bleeding into the brain). Chronic hypertension can damage blood vessels in the brain, leading to the formation of blood clots or weakening of vessel walls, increasing the risk of stroke.
E. Chronic hypertension is a significant risk factor for the development of atrial fibrillation, a type of irregular heart rhythm characterized by rapid and chaotic electrical activity in the upper chambers of the heart (atria). The structural changes in the heart caused by hypertension, such as left atrial enlargement and fibrosis, can predispose individuals to atrial fibrillation.
D. Familial hypercholesterolemia is a genetic disorder characterized by high levels of LDL cholesterol in the blood. While chronic hypertension itself does not directly cause familial hypercholesterolemia, hypertension and hypercholesterolemia are often comorbid conditions that contribute to cardiovascular risk. Both conditions can lead to atherosclerosis, increasing the risk of heart disease and stroke.
F. Isolated systolic hypertension refers to elevated systolic blood pressure with normal diastolic blood pressure. While isolated systolic hypertension itself is a manifestation of hypertension, it is not a direct result of end-organ damage. However, if left untreated, isolated systolic hypertension can lead to complications such as stroke, heart failure, and kidney disease, which are consequences of end-organ damage from chronic hypertension.
Correct Answer is C
Explanation
C Dyspnea, or difficulty breathing, is a classic symptom of circulatory overload. When the circulatory system is overloaded with excess fluid, it can lead to pulmonary congestion and impaired gas exchange, resulting in dyspnea. Patients experiencing circulatory overload may exhibit signs such as shortness of breath, increased respiratory rate, and crackles on lung auscultation.
A Flushing, or the sudden reddening of the skin, is not typically associated with circulatory overload. Instead, it can be a symptom of various conditions such as fever, allergic reactions, or hormonal changes. While circulatory overload may lead to fluid retention and increased blood volume, flushing is not a characteristic symptom.
B Vomiting is also not a typical symptom of circulatory overload. Vomiting can be caused by various factors such as anesthesia, pain medications, or postoperative ileus. While fluid overload can result in gastrointestinal symptoms like nausea and abdominal discomfort, vomiting is not a specific indicator of circulatory overload.
D Bradycardia, or a slow heart rate, is not typically associated with circulatory overload. Instead, it can be caused by factors such as medications (e.g., opioids, beta-blockers), vagal stimulation, or underlying cardiac conditions. In circulatory overload, the body's compensatory response is often tachycardia (increased heart rate) rather than bradycardia.
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