What are the risk factors associated with peptic ulcer disease?
(Select All that Apply.)
Family history
Blood type A
Acetaminophen (Tylenol) intake for pain
Smoking tobacco
Drinking caffeine
Correct Answer : A,D,E
A. Family history: Having a family history of peptic ulcers increases the risk of developing the condition, suggesting a genetic predisposition.
B. Blood type A: There is no direct association between blood type A and peptic ulcer disease.
C. Acetaminophen (Tylenol) intake for pain: Acetaminophen is generally considered safe for pain relief and is not a significant risk factor for peptic ulcer disease. However, nonsteroidal anti-inflammatory drugs (NSAIDs) such as aspirin, ibuprofen, and naproxen are known to increase the risk of peptic ulcers.
D. Smoking tobacco: Smoking tobacco is a significant risk factor for peptic ulcer disease. Tobacco use increases stomach acid production, weakens the protective lining of the stomach and duodenum, and impairs the healing of ulcers.
E. Drinking caffeine: While caffeine consumption alone may not directly cause peptic ulcers, excessive intake of caffeinated beverages such as coffee, tea, and soda can aggravate existing ulcers by stimulating stomach acid production and increasing gastric acidity.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A) Theophylline:
Theophylline is a bronchodilator medication that is used for the long-term management of asthma and chronic obstructive pulmonary disease (COPD). It has a slower onset of action compared to short-acting beta-agonists like albuterol and is not typically used as the initial treatment for acute asthma exacerbations.
B) Montelukast:
Montelukast is a leukotriene receptor antagonist used for the long-term control and prevention of asthma symptoms, but it is not typically used as the initial treatment for acute asthma exacerbations. Its onset of action is slower compared to short-acting beta-agonists like albuterol.
C) Albuterol.
Albuterol is a short-acting beta agonist bronchodilator commonly used as a first-line treatment for acute asthma exacerbations. It works by quickly relaxing the smooth muscles of the airways, leading to rapid bronchodilation and improvement of airflow. This can help alleviate the patient's symptoms of shortness of breath and wheezing.
D) Salmeterol:
Salmeterol is a long-acting beta-agonist bronchodilator used for the maintenance treatment of asthma and COPD, but it is not typically used as the initial treatment for acute asthma exacerbations. Its onset of action is slower compared to short-acting beta-agonists like albuterol, and it is not recommended for rapid relief of acute symptoms.
Correct Answer is C
Explanation
A. Decreased cardiac output: Inotropic medications are intended to improve cardiac function and increase cardiac output, so monitoring for signs of decreased cardiac output would be contrary to the expected therapeutic effect of these medications.
B. Increased afterload: Inotropic medications primarily affect the contractility of the heart muscle and do not typically have a direct effect on afterload (the force against which the heart must pump blood). While changes in afterload can occur as a secondary consequence of altered cardiac function, monitoring for signs of increased afterload would not be the primary focus after administering an inotropic medication.
C. Increased cardiac output.
Inotropic medications are drugs that affect the contractility of the heart muscle. They are often used in the management of shock to improve cardiac function and increase cardiac output. Therefore, after administering an inotropic medication, the nurse would monitor the patient for signs of increased cardiac output, such as improved peripheral perfusion, increased blood pressure, and resolution of signs and symptoms of shock.
D. Slowing of the heart rate: Inotropic medications can affect heart rate indirectly by altering cardiac output, but their primary effect is on contractility rather than heart rate. Monitoring for signs of bradycardia (slowing of the heart rate) may be appropriate in certain clinical situations, but it is not the primary consideration after administering an inotropic medication for shock.
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