This type of hypersensitivity reaction involves failure of the development of self-tolerance:
delayed hypersensitivity reaction
antibody-mediated reaction
autoimmune reaction
immediate hypersensitivity reaction
The Correct Answer is C
A. Delayed hypersensitivity reaction: This type of hypersensitivity reaction, also known as Type IV hypersensitivity, involves a delayed response by the immune system and is mediated by T cells. It does not necessarily involve a failure of self-tolerance.
B. Antibody-mediated reaction: This type of hypersensitivity reaction, also known as Type II hypersensitivity, involves antibodies targeting specific antigens on cell surfaces, leading to cell destruction. It doesn't directly indicate a failure of self-tolerance but rather an immune response against specific cells or tissues.
C. Autoimmune reaction: In autoimmune reactions, the immune system fails to recognize certain body tissues or substances as "self," leading to an immune response against the body's own cells or tissues. This results from a breakdown in self-tolerance, where the immune system mistakenly targets its own body.
D. Immediate hypersensitivity reaction: This type of hypersensitivity reaction, also known as Type I hypersensitivity, involves an immediate response by the immune system to an allergen. It includes conditions like allergies and anaphylaxis, where the immune system reacts strongly to substances that are normally harmless. This response does not necessarily indicate a failure of self-tolerance but rather an exaggerated response to specific antigens.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. "Tetracycline inhibits protein synthesis."
Explanation: Tetracycline antibiotics interfere with bacterial protein synthesis by binding to the bacterial ribosomes. This binding prevents the attachment of transfer RNA (tRNA) to the messenger RNA (mRNA) complex, effectively inhibiting the production of proteins that are crucial for bacterial growth and replication.
B. "Tetracycline blocks RNA synthesis."
Explanation: This statement is incorrect. Tetracycline primarily affects protein synthesis, not RNA synthesis. It doesn't block the creation of RNA molecules in bacteria.
C. "Tetracycline degrades the bacterial cell wall."
Explanation: This statement is incorrect. Tetracycline antibiotics do not target bacterial cell walls. Agents like penicillins and cephalosporins are examples of antibiotics that disrupt bacterial cell walls.
D. "Tetracycline binds to magnesium ions."
Explanation: This statement is incorrect. Tetracycline does bind to certain metal ions, but it's not primarily through magnesium ions. The binding to bacterial ribosomes is a key mechanism of action for tetracyclines.
Correct Answer is C
Explanation
A. "Cephalosporins are assigned to generations based on their relative costs to administer."
This statement is incorrect. The classification of cephalosporins into generations is based on their antimicrobial spectrum and activity against specific bacteria, not their cost.
B. "Later generations of cephalosporins have lower resistance to destruction by beta-lactamases."
This statement is not accurate. In fact, later generations of cephalosporins have increased resistance to destruction by beta-lactamases, which are enzymes produced by bacteria that can break down certain antibiotics.
C. "Cephalosporins have increased activity against gram-negative bacteria with each new generation."
This statement is correct. Cephalosporins are grouped into generations (first to fifth) based on their antibacterial spectrum. As the generations progress, there is an increase in activity against gram-negative bacteria, among other improvements in their spectrum.
D. "First-generation cephalosporins have better penetration of the cerebrospinal fluid."
This statement is generally true. First-generation cephalosporins have better penetration into the cerebrospinal fluid, making them useful for treating certain central nervous system infections.
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