Aplastic anemia is a disorder in which the bone marrow cells that develop into mature blood cells become damaged. The damaged bone marrow cells can lead to low numbers of red blood cells, white blood cells, and/or platelets. When the cause of aplastic anemia cannot be diagnosed, the cause is likely which of the following?
Radiation exposure
An autoimmune disorder
Chemotherapy drugs
Toxins
The Correct Answer is B
A. Radiation exposure: Radiation exposure can cause aplastic anemia, but if the cause is unknown, it is less likely to be due to a specific and identifiable event like radiation exposure.
B. An autoimmune disorder: When the cause of aplastic anemia cannot be diagnosed, it is often attributed to an autoimmune disorder where the body attacks its own bone marrow cells.
C. Chemotherapy drugs: Chemotherapy drugs can cause aplastic anemia, but this cause would typically be known based on the patient's medical history.
D. Toxins: Toxins can cause aplastic anemia, but like chemotherapy drugs and radiation, this cause would generally be identifiable.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. "Do you sleep well at night?" Sleep disturbances, including insomnia and night sweats, are common symptoms of menopause.
B. "Have you experienced increased hair growth?" Increased hair growth is not typically associated with menopause; rather, menopause is often associated with hair thinning or loss.
C. "Have you been experiencing chills?" Chills are not a common symptom of menopause; hot flashes and night sweats are more typical.
D. "When did you begin your menses?" This question is not relevant to confirming menopause symptoms, as it focuses on the onset of menstruation rather than menopausal changes.
Correct Answer is C
Explanation
A. DIC is not characterized by decreased production of clotting factors. Instead, it involves consumption of clotting factors due to widespread activation of coagulation.
B. While platelet dysfunction may occur in DIC due to consumption and depletion of platelets, the primary pathophysiological process in DIC is the activation of clotting factors and formation of blood clots throughout the vasculature.
C. Excessive activation of clotting factors and widespread clot formation is the primary pathophysiological process in DIC. This leads to the formation of numerous small blood clots throughout the body's blood vessels, resulting in organ dysfunction and potentially life-threatening complications.
D. Excessive platelet production is not a characteristic feature of DIC. Instead, platelets are consumed and depleted as they are incorporated into the blood clots formed during the
coagulation cascade in DIC.
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