Choice A rationale

Administering appropriate antibiotic therapy is a common nursing intervention to manage H. pylori infection. The recommended treatment for H. pylori typically involves using a triple-

drug therapy regimen, including a proton pump inhibitor or PPI like omeprazole, and two antibiotics, which include clarithromycin plus either metronidazole or amoxicillin. This treatment helps eradicate the bacterial infection through different mechanisms of action.

Choice B rationale

While maintaining a high-fiber diet is generally beneficial for overall health, it is not specifically related to the management of H. pylori infection. The primary treatment for H. pylori is antibiotic therapy, not dietary changes.

Choice C rationale

Over-the-counter antacids can help to neutralize stomach acid and provide temporary relief from symptoms, but they do not treat the underlying H. pylori infection. Therefore, while they may be used as part of symptom management, they are not a primary treatment strategy.

Choice D rationale

Assisting the patient with proper hand hygiene is always important in healthcare settings to prevent the spread of infections. However, it is not specifically related to the management of H. pylori infection. The bacteria are typically contracted during childhood, and the exact modes of transmission are not fully understood.

Choice A rationale
The UREA breath test is used to detect Helicobacter pylori (H. pylori), a type of bacteria that may infect the stomach and is a main cause of ulcers in both the stomach and duodenum (the first part of the small intestine).
H. pylori produces an enzyme called urease, which breaks urea down into ammonia and carbon dioxide. During the test, a tablet containing urea is swallowed and the amount of exhaled carbon dioxide is measured. This indicates the presence of H. pylori in the stomach. Therefore, the UREA breath test measures urea levels to determine if H. pylori is present.

Choice B rationale
While it is true that the presence of Helicobacter pylori antibodies in the blood can indicate an
H. pylori infection, this is typically detected using a blood test, not a UREA breath test. The UREA breath test specifically measures the amount of carbon dioxide in your breath after you drink a special solution containing urea.
H. pylori bacteria break down urea into carbon dioxide, and excess carbon dioxide in your breath is a sign of an H. pylori infection. Therefore, while Helicobacter pylori antibodies can indicate an H. pylori infection, they are not the lab value that the UREA breath test measures to determine if H. pylori is present.
Choice C rationale
Gastrin levels are not measured in a UREA breath test. Gastrin is a hormone that stimulates the production of gastric acid, which helps the stomach digest food. While gastrin levels can be elevated in individuals with certain conditions, such as gastrinomas or Zollinger-Ellison syndrome, they are not directly related to H. pylori infections or measured in a UREA breath test.
Choice D rationale
Pepsinogen levels are not measured in a UREA breath test. Pepsinogen is a precursor enzyme (zymogen) produced by the gastric chief cells, and it is converted to the enzyme pepsin in the presence of stomach acid. While pepsinogen levels can be used to assess certain conditions, such as atrophic gastritis, they are not directly related to H. pylori infections or measured in a UREA breath test.