A client with chronic cirrhosis has esophageal varices.
It is most important for the nurse to monitor the client for the onset of which problem?
Anorexia
Brown, foamy urine.
Clay-colored stool
Hematemesis
The Correct Answer is D
Choice A rationale:
Anorexia is a common symptom of cirrhosis, but it is not the most immediate concern in a client with esophageal varices.
Anorexia can result from various factors, including impaired liver function, hormonal imbalances, early satiety due to ascites,
and nausea.
While it's important to address anorexia to maintain nutritional status, it doesn't directly indicate a life-threatening
complication like bleeding varices.
Choice B rationale:
Brown, foamy urine is a characteristic of kidney problems, specifically proteinuria.
It's not directly associated with esophageal varices or their complications.
While cirrhosis can affect kidney function due to fluid imbalances and impaired filtering, brown, foamy urine wouldn't be the
primary indicator of bleeding varices.
Choice C rationale:
Clay-colored stool is a sign of biliary obstruction, which can occur in cirrhosis due to impaired bile flow.
However, it's not a direct indicator of bleeding varices.
Clay-colored stool results from a lack of bile pigments in the stool, which give it its typical brown color.
Choice D rationale:
Hematemesis, or vomiting of blood, is the most critical symptom to monitor in a client with esophageal varices.
Here's why:
Esophageal varices are enlarged, swollen veins in the esophagus that develop due to increased pressure in the portal venous
system, a common consequence of cirrhosis.
These varices are fragile and prone to rupture, which can lead to massive, life-threatening bleeding.
Hematemesis is a clear sign that variceal bleeding has occurred and requires immediate intervention.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A rationale:
Famotidine is a histamine-2 receptor antagonist (H2RA) that decreases gastric acid secretion. It is often used to treat conditions such as ulcers, gastroesophageal reflux disease (GERD), and heartburn.
Bismuth subsalicylate, metronidazole, tetracycline, and pantoprazole are all medications used in the treatment of H. pylori infection. However, these medications can interact with famotidine, potentially leading to decreased efficacy or increased side effects.
Therefore, it is important for the nurse to review the client's electronic medical record for any current or recent use of famotidine before administering the H. pylori treatment regimen. This will help to ensure the safety and effectiveness of the treatment.
Choice B rationale:
Loperamide is an antidiarrheal medication. It is not typically used in the treatment of H. pylori infection and is not known to interact with the medications used in this regimen.
Choice C rationale:
Aspirin is a salicylate medication that is often used to treat pain, fever, and inflammation. It can also be used to prevent blood clots. However, aspirin can interact with bismuth subsalicylate, potentially leading to increased risk of bleeding.
Therefore, it is important for the nurse to assess the client's risk of bleeding before administering the H. pylori treatment regimen if they are also taking aspirin.
Choice D rationale:
Ipratropium is a bronchodilator medication that is used to treat asthma and chronic obstructive pulmonary disease (COPD). It is not typically used in the treatment of H. pylori infection and is not known to interact with the medications used in this regimen.
Correct Answer is A
Explanation
Choice A rationale:
Lactulose is a non-absorbable disaccharide that is metabolized by bacteria in the colon to produce lactic acid and acetic acid. These acids acidify the colon, which traps ammonia (NH3) in the colon and converts it to ammonium (NH4+). Ammonium is not absorbed by the colon and is excreted in the stool. This ammonia-lowering effect is the primary mechanism by which lactulose improves mental status in patients with hepatic encephalopathy.
Hepatic encephalopathy is a neuropsychiatric syndrome that occurs in patients with severe liver disease. It is caused by the buildup of ammonia in the blood, which can cross the blood-brain barrier and cause cerebral edema and impaired brain function.
The clinical manifestations of hepatic encephalopathy range from mild confusion and personality changes to coma. Improved mental status is a key therapeutic goal in the treatment of hepatic encephalopathy.
Studies have shown that lactulose can improve mental status in patients with hepatic encephalopathy. A meta-analysis of 11 randomized controlled trials found that lactulose was associated with a significant improvement in mental status compared to placebo.
The improvement in mental status is typically seen within 24 to 48 hours of starting lactulose therapy.
Choice B rationale:
Increased urine output is not a direct therapeutic effect of lactulose. Lactulose can cause diarrhea, which can lead to increased urine output due to fluid loss. However, this is not the primary mechanism by which lactulose improves mental status in patients with hepatic encephalopathy.
Choice C rationale:
Reduction in the number of liquid stools is a common side effect of lactulose. However, it is not a therapeutic goal in the treatment of hepatic encephalopathy. The goal of lactulose therapy is to improve mental status by lowering ammonia levels in the blood.
Choice D rationale:
Ability to ambulate independently is not a direct therapeutic effect of lactulose. Lactulose can improve mental status, which may indirectly lead to improved ambulation in some patients. However, this is not the primary mechanism by which lactulose works.
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