Which is a major side effect of radioactive iodine treatment for hyperthyroidism?
TSH low,T3 & T4 elevated
TSH elevated, T3 & T4 low
TSH,T3 & T4 low
TSH, T4 elevated and T3 low
The Correct Answer is B
Choice A rationale: Elevated T3 and T4 with low TSH levels are indicative of hyperthyroidism, not a side effect of radioactive iodine treatment.
Choice B rationale: One of the expected side effects of RAI treatment for hyperthyroidism is permanent hypothyroidism (or underactive thyroid, when the thyroid does not produce enough hormones). Elevated TSH levels with low T3 and T4 are indicative of hypothyroidism.
Choice C rationale: Low TSH, T3, and T4 levels are not typical outcomes of radioactive iodine treatment; typically, the treatment leads to a decrease in T3 and T4 with increased TSH levels.
Choice D rationale: Elevated TSH and low T3 is major side effect of radioactive iodine treatment for hyperthyroidism. However, it does not cause elevated T4 levels.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale: Intubation and mechanical ventilation may be needed if the respiratory muscles are affected by GBS, but this is not a routine intervention at this stage.
Choice B rationale: One of the main treatments of GBS is IV immunoglobulin therapy (IVIG), which involves infusing antibodies from donated blood plasma to block the harmful immune response that damages the nerves. Sandoglobulin is one of the brand names of IVIG used for GBS. IVIG can help shorten the duration and severity of the disease, and improve the recovery rate.
Choice C rationale: Methylprednisolone is a corticosteroid that can reduce inflammation, but it is not recommended for GBS, as it may worsen the condition or increase the risk of infection.
Choice D rationale: NG feeding tube may be required if the patient has difficulty swallowing, but this is also not a standard intervention at this time.
Correct Answer is C
Explanation
Choice A rationale: The patient with type 2 diabetes admitted with pneumonia might not necessarily need insulin therapy to prevent ketoacidosis.
Choice B rationale: Type 2 diabetes doesn't typically involve islet cell antibodies destroying the pancreas's ability to produce insulin.
Choice C rationale: In this scenario, the patient might have enough endogenous insulin to prevent ketosis but could be at risk for hyperosmolar hyperglycemic syndrome due to illness-induced stress.
Choice D rationale: Type 2 diabetes often involves some degree of endogenous insulin secretion, and not all patients require daily insulin injections.
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