The nurse is caring for a patient in the Intensive Care Unit (ICU) with a ventriculostomy and intracranial pressure (ICP) reading of 30mmHg. The nurse notes a widened pulse pressure, irregular respirations. and bradycardia. The neurosurgeon instructs the nurse to drain cerebrospinal fluid. The nurse would also anticipate which medication administration order from the provider?
3% Normal Saline
Nitroprusside
Norepinephrine
Furosemide
The Correct Answer is A
A) 3% Normal Saline:
Elevated ICP can lead to a life-threatening condition known as cerebral herniation, and it often triggers the Cushing’s triad—widened pulse pressure, bradycardia, and irregular respirations, all of which the nurse has noted. Hypertonic saline (such as 3% Normal Saline) is commonly used to manage increased ICP because it creates an osmotic gradient that draws fluid out of the brain tissue, reducing cerebral edema and lowering ICP. The goal is to reduce swelling and prevent further brain damage.
B) Nitroprusside:
Nitroprusside is a potent vasodilator that is used to treat hypertension and reduce afterload in conditions like heart failure or aortic dissection. However, in this case, nitroprusside is contraindicated because it could cause a drop in systemic blood pressure, which would worsen cerebral perfusion in a patient with elevated ICP.
C) Norepinephrine:
Norepinephrine is a vasopressor used to treat hypotension by increasing systemic vascular resistance and blood pressure. Although norepinephrine may be used to support blood pressure in critically ill patients, in this context of elevated ICP, the priority is to reduce ICP, not to increase blood pressure further. Increasing the systemic vascular resistance with norepinephrine could exacerbate ICP by increasing the volume of blood in the cranium
D) Furosemide:
Furosemide is a loop diuretic that can reduce fluid volume and help treat conditions like edema or heart failure. While furosemide can reduce overall fluid volume, it is not typically used to manage elevated ICP in this context. Diuretics like furosemide may have a role in managing cerebral edema, but they are not as effective as hypertonic saline in rapidly reducing ICP.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["D","E"]
Explanation
A) Pneumonia: Pneumonia is a direct cause of ARDS, as it involves direct inflammation and infection of the lung tissue, leading to impaired oxygen exchange and damage to the alveolar-capillary membrane. It results in fluid accumulation in the lungs, which is a key characteristic of ARDS.
B) Drowning: Drowning is another direct cause of ARDS. It involves the aspiration of water into the lungs, which directly damages lung tissue, causing pulmonary edema and impaired oxygenation.
C) Aspiration: Aspiration of food, liquid, or vomit into the lungs is also a direct cause of ARDS. The aspirated material can lead to chemical pneumonia, bacterial infection, and inflammation of the lungs, which in turn causes ARDS.
D) Sepsis: Sepsis is an indirect cause of ARDS. It can lead to widespread inflammation throughout the body, including the lungs, through the release of inflammatory mediators (cytokines, interleukins, etc.). These systemic inflammatory responses can increase capillary permeability in the lungs, causing fluid to leak into the alveoli, which leads to ARDS.
E) Blood Transfusion: Blood transfusion, particularly when associated with transfusion-related acute lung injury (TRALI), is an indirect cause of ARDS. TRALI is a serious complication that can result from receiving blood products, where the transfused blood causes an immune response leading to pulmonary damage. It indirectly triggers inflammation and fluid buildup in the lungs, contributing to ARDS.
Correct Answer is D
Explanation
A) Prolonged prothrombin time (PT):
A prolonged PT is a sign of impaired coagulation, which is common in DIC as the clotting factors are consumed. In DIC, both clotting and bleeding occur, leading to prolonged PT. Therefore, a prolonged PT does not reflect a positive outcome. A positive outcome would show normalization or improvement of the clotting profile.
B) Elevated fibrinogen level:
Fibrinogen is often decreased in DIC because it is consumed in the process of forming clots. An elevated fibrinogen level would not indicate a positive outcome in DIC. Instead, fibrinogen levels typically fall in DIC due to widespread clotting activity. A positive outcome would involve a normalization or increase in fibrinogen levels as the condition improves.
C) Decreased platelet count:
Platelet count typically decreases in DIC because platelets are used up in the formation of clots, leading to thrombocytopenia. A decreased platelet count reflects ongoing consumption of platelets and would not be a positive indicator of improvement. In a positive outcome, the platelet count would increase toward normal levels.
D) Decreased D-dimer level:
D-dimer is a product of fibrin degradation and is elevated in conditions like DIC, where abnormal clotting and fibrinolysis are occurring. A decreased D-dimer level indicates that the coagulation process is resolving, and fibrin degradation is returning to normal, reflecting a positive outcome. Monitoring the D-dimer level is a key indicator of improvement in DIC.
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