In the treatment of angina, the mechanism of action of an organic nitrate is:
Increased contractility and heart rate.
Slowed heart rate and decreased contractility of the heart.
Relaxation of arterial and venous smooth muscle.
Increased peripheral resistance.
The Correct Answer is C
A. Organic nitrates do not increase contractility and heart rate; they typically decrease myocardial oxygen demand.
B. Organic nitrates do not slow heart rate or decrease contractility of the heart.
C. The mechanism of action of organic nitrates in the treatment of angina is the relaxation of arterial and venous smooth muscle, leading to vasodilation and decreased myocardial oxygen demand.
D. Organic nitrates do not increase peripheral resistance; they typically cause vasodilation.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Explanation:
A. Noncompliance implies that the client is not following a prescribed treatment plan, which may not be the case here as the client is being educated.
B. Deficient knowledge reflects the need for education regarding lifestyle changes, such as dietary modifications, which is appropriate for a client newly diagnosed with hyperlipidemia.
C. Impaired health maintenance might be applicable if the client was not adhering to prescribed drug therapy, but this diagnosis doesn't address the need for education.
D. Self-care deficit may apply if the client is unable to perform necessary self-care activities due to physical limitations but does not address the educational aspect of dietary changes.
Correct Answer is B
Explanation
A. Aspirin is not primarily used to inhibit peptic ulcer disease; it is used to prevent cardiovascular events by decreasing platelet aggregation.
B. Aspirin decreases platelet aggregation, making it useful in preventing clot formation in conditions such as angina and myocardial infarction.
C. Aspirin is not used to reduce emphysema; it is primarily used for its antiplatelet effects.
D. Aspirin does not directly decrease carbon dioxide retention; its primary mechanism of action in cardiovascular disease is through antiplatelet effects.
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