Alzheimer's disease (AD)
manifests as neurofibrillary tangles and amyloid plaques that cause a disruption of brain impulses.
is an inherited disorder that, most often, begins before the age of fifty
can be caused by increased cerebral levels of acetylcholine
causes unusual sensory sensations such as numbness and shooting pain (paresthesia)
The Correct Answer is A
A. Manifests as neurofibrillary tangles and amyloid plaques that cause a disruption of brain impulses: Alzheimer's disease is characterized by the accumulation of amyloid plaques and neurofibrillary tangles in the brain, leading to disrupted neural communication.
B. Is an inherited disorder that, most often, begins before the age of fifty: While there are inherited forms of Alzheimer's (early-onset), most cases are sporadic and occur after age 65.
C. Can be caused by increased cerebral levels of acetylcholine: Alzheimer's disease is associated with decreased levels of acetylcholine, not increased levels.
D. Causes unusual sensory sensations such as numbness and shooting pain (paresthesia): Paresthesia is not a typical symptom of Alzheimer's disease; it is more related to neurological or peripheral nerve disorders.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. Steroid injections will be administered daily: This is incorrect; steroid therapy may exacerbate Cushing's disease. The condition is often due to excessive production of cortisol, not a deficiency.
B. Weight gain and edema are present: Hyperaldosteronism, often associated with Cushing's syndrome, leads to sodium and water retention, resulting in weight gain and edema.
C. Painful leg cramps are common from hyperkalemia: Hyperaldosteronism typically causes hypokalemia, not hyperkalemia, leading to muscle cramps, not leg pain from hyperkalemia.
D. Decreased amount of body hair is frequently seen: Cushing's disease often leads to hirsutism (increased body hair), not a decrease.
Correct Answer is A
Explanation
A. Diminished serum albumin levels cause water to shift from blood to tissue: In cirrhosis, liver dysfunction leads to decreased production of albumin, a protein that helps maintain oncotic pressure. Low albumin levels cause fluid to shift from the vascular space into the tissues, resulting in ascites and peripheral edema.
B. Portal hypotension causes a fluid shift from the abdominal cavity into the portal veins: Portal hypertension, not hypotension, is a common feature of cirrhosis, but it leads to ascites by increasing pressure in the portal venous system, not by shifting fluid into the portal veins.
C. Hypoaldosteronism causes a fluid volume deficit: shifting water from blood into tissue: Cirrhosis often leads to hyperaldosteronism, not hypoaldosteronism, resulting in sodium and water retention, which contributes to edema.
D. Aberrations of the portal system cause a back-up of blood that leads to hydronephrosis: Hydronephrosis is related to obstruction of the urinary tract, not a complication of portal hypertension or cirrhosis.
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