A patient has had chronic kidney disease (CKD) for many years. The patient sustains a hip fracture. The high risk of fractures is related to a(n)
inability of the kidneys to produce erythropoietin.
increased ability to absorb calcium from the foods that are eaten.
decreased ability of the kidneys to activate vitamin D.
decreased serum phosphate.
The Correct Answer is C
A. While CKD does reduce erythropoietin production, this affects anemia rather than bone health or fracture risk.
B. CKD often leads to decreased calcium absorption due to impaired vitamin D activation, not an increased absorption. C. In CKD, the kidneys lose the ability to convert vitamin D to its active form, impairing calcium absorption from the intestine and weakening
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. While sodium reduction might indirectly impact weight in some cases, its primary role in blood pressure control is related to its effect on blood volume and vascular resistance.
B. Sodium reduction is proven to help reduce blood pressure by lowering blood volume and decreasing vascular resistance.
C. Sodium actually leads to fluid retention, which can increase blood volume and raise blood pressure, not decrease it.
D. There is no direct link between sodium intake and smoking habits; reducing sodium primarily affects blood pressure management independently of smoking.
Correct Answer is D
Explanation
A. Atherosclerosis typically leads to vessel narrowing, but it does not specifically describe the formation of an aneurysm, which involves wall weakening and outpouching rather than just narrowing.
B. While a blood clot can be a complication of an AAA, it is not the primary cause of aneurysm formation. An AAA results from wall weakness rather than a clot.
C. Streptococcal bacteria are not related to the development of abdominal aortic aneurysms; aneurysms are associated with chronic conditions affecting the vascular wall, not bacterial infections.
D. Chronic hypertension contributes to the weakening of the arterial walls, leading to the formation of an aneurysm as the pressure causes the wall to bulge or outpouch. This correctly reflects the pathophysiology of AAA.
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