A nurse is providing discharge education to a client who has valvular heart disease and peripheral vascular disease. Which of the following should the nurse include in the teaching about valvular heart disease?
Comply with compression therapy.
Monitor wounds on lower extremities.
Take antiplatelet medications as ordered.
Monitor for gradual onset of symptoms
The Correct Answer is D
A. Comply with compression therapy: Compression therapy is commonly used in the management of peripheral vascular disease (PVD), not valvular heart disease. It involves applying external pressure to the limbs to improve circulation and reduce edema. While this intervention may be relevant for a client with PVD, it is not specific to valvular heart disease.
B. Monitor wounds on lower extremities: Monitoring wounds on lower extremities is important for clients with peripheral vascular disease (PVD) to assess for signs of poor wound healing, infection, or tissue ischemia. However, it is not directly related to valvular heart disease. Therefore, while wound monitoring may be appropriate for this client, it is not specific to valvular heart disease education.
C. Take antiplatelet medications as ordered: Antiplatelet medications, such as aspirin or clopidogrel, are commonly prescribed for clients with peripheral vascular disease (PVD) to reduce the risk of thrombotic events and improve blood flow. However, they are not typically indicated as a primary treatment for valvular heart disease. While some clients with valvular heart disease may have comorbidities that warrant antiplatelet therapy, it is not specific to valvular heart disease education.
D. Valvular heart disease involves dysfunction of one or more heart valves, leading to impaired blood flow within the heart. The nurse should educate the client on monitoring for the gradual onset of symptoms related to valvular heart disease. These symptoms may include dyspnea (shortness of breath), fatigue, palpitations, chest discomfort, and edema. Monitoring for these symptoms allows for early detection of disease progression or exacerbation, prompting timely intervention and management.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A) Hypoxia and acidosis While hypoxia and acidosis are serious complications of shock, they are physiological rather than psychological outcomes. These conditions primarily affect the body's metabolic and respiratory functions, rather than mental health or behavior.
B) Hearing deficits and increased risk of glaucoma Hearing deficits and increased risk of glaucoma are potential complications associated with certain medical conditions or medications but are not directly related to the psychological outcomes of shock. These conditions affect sensory perception (hearing and vision) rather than mental health or behavior.
C) Bipolar behaviors and schizotypal behaviors Bipolar behaviors and schizotypal behaviors are manifestations of mood and psychotic disorders, respectively, and are not typical adverse outcomes of shock. While psychological disturbances can occur in critically ill patients, they are not commonly characterized by specific psychiatric diagnoses like bipolar or schizotypal behaviors.
D) Disorientation and depression Disorientation and depression are common adverse psychological outcomes experienced by patients who have spent an extended period in the intensive care unit (ICU) due to complications related to shock. Prolonged ICU stays, medical interventions, sedation, and physical discomfort can contribute to feelings of confusion, disorientation, and depression in patients. Therefore, it is essential for the nurse to educate the client about these potential psychological effects and provide appropriate support and resources to address them during the transition to the medical unit.
Correct Answer is C
Explanation
A. Tau protein: Tau protein is primarily associated with Alzheimer's disease and other tauopathies, not Lewy body dementia.
B. Neurofibrillary tangles: Neurofibrillary tangles are aggregates of hyperphosphorylated tau protein found in Alzheimer's disease, not typically in Lewy body dementia.
C. Alpha-synuclein protein: Lewy bodies, which are abnormal aggregates of alpha-synuclein protein, are a hallmark pathology of Lewy body dementia. These protein aggregates disrupt neuronal function and are responsible for the cognitive, motor, and emotional symptoms seen in Lewy body dementia.
D. Beta-amyloid protein: Beta-amyloid protein is primarily associated with Alzheimer's disease, not Lewy body dementia. It forms plaques in the brain, which contribute to neurodegeneration and cognitive decline in Alzheimer's disease.
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