A nurse is assessing a client who reports hearing loss. Which of the following statements indicates that the hearing loss is affecting the client's ability to perform activities of daily living (ADLs)?

A. "Your heart condition is caused by excessive stretching of the ventricles": Dilated cardiomyopathy (DCM) is characterized by the dilation or enlargement of the heart's chambers, particularly the ventricles. This dilation results in weakened myocardial contraction and impaired systolic function. The primary cause of DCM is often idiopathic (unknown), but it can also be secondary to various factors such as genetics, infections, toxins, or systemic diseases. The statement correctly identifies the pathophysiological basis of DCM as excessive stretching or dilation of the ventricles, leading to impaired cardiac function.

B. "Your heart condition is caused when the ventricular tissue becomes fibrous and fatty": This statement is incorrect. Fibrous and fatty changes in ventricular tissue are characteristic of another type of cardiomyopathy known as hypertrophic cardiomyopathy (HCM), not dilated cardiomyopathy (DCM). In DCM, the primary abnormality is dilation of the heart chambers rather than fibrous and fatty changes in the myocardium.

C. "Your heart condition is caused from stiffening of the walls of the ventricles": This statement describes restrictive cardiomyopathy (RCM), not dilated cardiomyopathy (DCM). In restrictive cardiomyopathy, the ventricular walls become stiff and rigid, impairing diastolic filling and ventricular relaxation. However, in DCM, the primary abnormality is dilation or enlargement of the ventricles rather than stiffening of the ventricular walls.

D. "Your heart condition is caused by thickening of the ventricular walls and septum": This statement describes hypertrophic cardiomyopathy (HCM), not dilated cardiomyopathy (DCM). In HCM, there is abnormal thickening of the ventricular walls and septum, leading to reduced chamber size and impaired diastolic filling. However, in DCM, the primary abnormality is dilation or enlargement of the ventricles rather than thickening of the ventricular walls.

A. Completing hourly endotracheal suctioning: Hourly endotracheal suctioning is not typically indicated for a client with increased intracranial pressure (ICP). Frequent suctioning can lead to increased intrathoracic pressure and potentially compromise venous return, which may further elevate ICP. Suctioning should be performed as needed to maintain airway patency while minimizing the risk of increasing ICP.

B. Ensuring proper ventriculostomy transducer levels: Ensuring proper ventriculostomy transducer levels is important for accurate measurement of intracranial pressure (ICP) but may not directly alleviate elevated ICP. Monitoring ICP through ventriculostomy allows for timely detection of changes in ICP, which can guide interventions to manage elevated pressure levels. However, it is not a direct intervention to reduce ICP.

C. Monitoring volume status: Monitoring volume status is important in managing a client with increased intracranial pressure (ICP) as both hypovolemia and hypervolemia can impact ICP. However, monitoring volume status alone does not directly address elevated ICP. Interventions to optimize volume status, such as fluid administration or diuresis, may be implemented based on assessment findings, but they should be done cautiously to avoid exacerbating cerebral edema or altering cerebral perfusion.

D. Elevating the head of the bed 15°: Elevating the head of the bed 15° (or higher) is a crucial intervention for managing a client with increased intracranial pressure (ICP). This position helps promote venous drainage from the brain, reducing venous congestion and intracranial pressure. Elevating the head of the bed also helps prevent cerebrospinal fluid (CSF) from pooling in the brain, which can further increase ICP. Placing the client in a semi-upright position is a standard practice in managing ICP and is recommended in various clinical guidelines.