A nurse is performing preoperative assessments on a client who has a suspected detached retina. Which of the following should the nurse expect to find?

Tonometer intraocular pressure reading 8 mm Hg

Smooth retina edges identified on slit-lamp biomicroscope examination

Visual acuity of 20/20 using the Snellen eye chart

Lens and cornea appear intact during ophthalmoscope exam

Answer and Explanation

The Correct Answer is A

Tonometer intraocular pressure reading 8 mm Hg (Option A): A low intraocular pressure reading may indicate a detached retina. In a detached retina, the vitreous humor (gel-like substance in the eye) may leak into the space behind the retina, causing a decrease in intraocular pressure. However, it's important to note that intraocular pressure alone cannot definitively diagnose a detached retina, and further diagnostic tests, such as a dilated eye examination or retinal imaging, would be needed for confirmation.

Smooth retina edges identified on slit-lamp biomicroscope examination (Option B): In the case of a detached retina, the edges of the retina may appear irregular or undulating rather than smooth. This irregularity is often observed during a dilated eye examination rather than with a slit-lamp biomicroscope.

Visual acuity of 20/20 using the Snellen eye chart (Option C): Visual acuity may be affected in a detached retina, depending on the extent and location of the detachment. However, visual acuity alone cannot confirm a detached retina, as other factors such as refractive errors or cataracts can also affect visual acuity.

Lens and cornea appear intact during ophthalmoscope exam (Option D): While a detached retina may be visualized during an ophthalmoscope examination as a gray or whitish area behind the lens, the appearance of the lens and cornea being intact does not definitively diagnose a detached retina. A thorough dilated eye examination by an ophthalmologist is necessary for accurate diagnosis.

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Correct Answer is B

Explanation

A. Decrease sedation: Decreasing sedation may help reduce intracranial pressure (ICP) by allowing the client to have a more responsive level of consciousness. However, it is not a direct treatment for brain herniation. Sedation reduction should be done cautiously to prevent agitation and further increases in ICP.

B. Hyperventilate the client: Hyperventilation is a temporary intervention used to reduce intracranial pressure by inducing cerebral vasoconstriction, which decreases cerebral blood flow and intracranial volume. However, it is typically reserved for acute situations and is not considered a definitive treatment for brain herniation. Prolonged or excessive hyperventilation can lead to cerebral ischemia and should be used cautiously.

C. Lower blood pressure: Lowering blood pressure may help reduce cerebral perfusion pressure, which can mitigate the risk of further brain injury during herniation. However, lowering blood pressure alone is not a direct treatment for brain herniation. It may be part of the overall management strategy to prevent secondary injury.

D. Reduce the temperature in the room: Reducing the temperature in the room, or therapeutic hypothermia, is sometimes used in the management of elevated intracranial pressure and brain injury. Lowering body temperature can reduce metabolic demands and cerebral edema, thereby lowering intracranial pressure. However, it is not a direct treatment for brain herniation and should be implemented cautiously to prevent complications such as shivering and hypotension.

A. Tau protein B. Neurofibrillary tangles C. Alpha-synuclein protein D. Beta-amyloid protein

Correct Answer is C

Explanation

A. Tau protein: Tau protein is primarily associated with Alzheimer's disease and other tauopathies, not Lewy body dementia.

B. Neurofibrillary tangles: Neurofibrillary tangles are aggregates of hyperphosphorylated tau protein found in Alzheimer's disease, not typically in Lewy body dementia.

C. Alpha-synuclein protein: Lewy bodies, which are abnormal aggregates of alpha-synuclein protein, are a hallmark pathology of Lewy body dementia. These protein aggregates disrupt neuronal function and are responsible for the cognitive, motor, and emotional symptoms seen in Lewy body dementia.

D. Beta-amyloid protein: Beta-amyloid protein is primarily associated with Alzheimer's disease, not Lewy body dementia. It forms plaques in the brain, which contribute to neurodegeneration and cognitive decline in Alzheimer's disease.

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