A nurse is caring for a client who recently had a streptococcal infection of the throat, and is now experiencing new-onset flank pain. Urinalysis is positive for proteinuria and hematuria. The client's urine output for the past 24 hours has been 250 mL, and BUN and creatinine are elevated. On assessment, the nurse notes pitting edema to the bilateral lower extremities. Which of the following interventions is appropriate in the plan of care?
Encourage frequent ambulation.
Obtain weight weekly.
Place the client on a low-protein diet.
Encourage increased fluid intake.
The Correct Answer is B
A. Frequent ambulation could exacerbate the edema and is not a priority in the acute management of this condition.
B. Obtaining weight weekly is an appropriate intervention to monitor for changes in fluid status and guide treatment decisions.
C. A low-protein diet is not a standard treatment for PSGN.
D. Encouraging increased fluid intake is not recommended due to the client's oliguria and risk of fluid overload
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Related Questions
Correct Answer is C
Explanation
A. Tolvaptan does not increase the glomerular filtration rate. It actually works by blocking the action of a hormone that causes the kidneys to retain water, leading to increased urine output.
B. While Tolvaptan can slow the progression of polycystic kidney disease (PKD) and delay the need for dialysis or transplant, it does not guarantee prevention of these outcomes.
C. Tolvaptan is used to slow the growth of kidney cysts in patients with PKD.
D. Hyponatremia is a common side effect of Tolvaptan due to increased water loss through urine. However, this is not the primary role.
Correct Answer is B
Explanation
A. Calcium gluconate does not directly affect the elimination of potassium from the body or its excretion in the stool. It does not act on the kidneys or gastrointestinal tract to remove potassium. Its primary role is to stabilize the cardiac membrane.
B. Calcium gluconate stabilizes the cardiac membrane and reduces the risk of dysrhythmias associated with elevated serum potassium levels. Hyperkalemia can increase the risk of cardiac arrhythmias, and calcium gluconate helps to counteract these effects by protecting the heart muscle.
C. Calcium gluconate does not cause potassium to shift from the blood into the cells. The shift of potassium into cells is typically induced by other treatments such as insulin and glucose or beta- agonists. Calcium gluconate does not have this effect.
D. Calcium gluconate does not increase serum potassium levels. It primarily serves to protect the heart from the effects of high potassium levels. It does not have a direct effect on the potassium level itself but helps manage the consequences of hyperkalemia.
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