A nurse is caring for a client at a 10-week prenatal visit. The provider diagnoses the client with a missed spontaneous abortion confirmed by ultrasound and the nurse is discussing the plan of care with the client.
Which of the following statements should the nurse make?

Choice A rationale

Administering a rubella immunization during pregnancy is contraindicated because the rubella vaccine is a live attenuated virus vaccine. There is a theoretical risk of viral transmission to the fetus, which could lead to congenital rubella syndrome. Vaccination should occur postpartum to protect future pregnancies.

Choice B rationale

Breastfeeding is not contraindicated after receiving the rubella vaccine. Although rubella vaccine virus can be excreted in breast milk, it generally does not cause clinical illness in the infant and passive transfer of antibodies may provide some protection to the infant.

Choice C rationale

A rubella titer of 1: indicates a low level of rubella antibodies, meaning the client is susceptible to rubella infection. A titer of less than 1: or 1: is generally considered non-immune, requiring vaccination to prevent congenital rubella syndrome in future pregnancies.

Choice D rationale

Repeating a rubella titer in the third trimester is not a standard practice for assessing susceptibility. Once susceptibility is determined, the primary intervention is postpartum vaccination. The initial titer result accurately reflects immune status and further testing during pregnancy is unnecessary.

Choice A rationale: Blood pressure readings below 160/110 mm Hg overnight indicate some level of blood pressure control, which is a positive sign in hypertensive pregnancy conditions. The goal is to maintain pressures below this threshold to reduce risk of end-organ damage. Stable or lower pressures reduce cerebral and placental ischemia risk. Therefore, resting well with controlled BP suggests no immediate worsening, indicating progression is stable at this point.

Choice B rationale: A decrease in headache intensity temporarily is a favorable clinical sign. Headache in preeclampsia is caused by cerebral edema and vasospasm, so improvement indicates less neurological irritation or pressure. However, this is a transient improvement and must be interpreted cautiously, but the reduction alone does not indicate a worsening condition, so it is not a marker of poor progression.

Choice C rationale: An increased headache intensity rating to 7/10 signals significant neurological involvement and increased cerebral irritation, typical of worsening preeclampsia or impending eclampsia. Severe headaches in pregnancy with hypertension indicate cerebral vasospasm or edema, which may lead to seizures if untreated. This is a critical sign requiring urgent intervention to prevent maternal and fetal morbidity.

Choice D rationale: Persistent visual disturbances such as seeing spots or flashes are neurological symptoms indicating retinal or cerebral involvement due to vasospasm, ischemia, or edema. These symptoms are common in severe preeclampsia and herald worsening disease. Visual symptoms result from endothelial dysfunction affecting cerebral and retinal vessels, requiring immediate evaluation to prevent progression to eclampsia.

Choice E rationale: Epigastric discomfort reflects stretching or ischemia of the liver capsule from hepatic involvement in severe preeclampsia or HELLP syndrome. This pain typically presents as right upper quadrant or epigastric pain due to hepatocellular injury or microvascular thrombosis. It is a warning sign of multisystem involvement and potential progression to life-threatening complications such as hepatic rupture.

Choice F rationale: Hyperactive deep tendon reflexes (3+ to 4+) and positive clonus are clinical signs of central nervous system irritability caused by increased excitability of motor neurons. This occurs due to cerebral vasospasm and ischemia in severe preeclampsia and predicts risk for seizures (eclampsia). These neurological signs are crucial in assessing disease severity and necessitate urgent management.

Choice G rationale: Urine output between 25 and 55 mL/hr approaches the lower limit of normal (normal ≥30 mL/hr). Reduced urine output in preeclampsia indicates renal hypoperfusion or injury due to endothelial dysfunction and vasospasm, which can progress to acute kidney injury. Monitoring urine output is essential as oliguria signals worsening renal compromise, increasing maternal and fetal risk.