Which of the following is a true statement
Hypoventilation causes hypocapnia
Hyperventilation causes hypercapnia
Hyperventilation causes hypocapnia
Hyperventilation results in an increased PaCO2
The Correct Answer is C
Hyperventilation refers to an increased rate and depth of breathing, which results in excessive elimination of carbon dioxide (CO2) from the body. Carbon dioxide is an acidic gas that helps regulate the pH of the blood. When too much CO2 is lost through hyperventilation, it causes a decrease in the partial pressure of carbon dioxide in the blood (PaCO2), leading to a condition called hypocapnia.
Hypocapnia can cause symptoms such as lightheadedness, dizziness, and tingling in the fingers and toes.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"dropdown-group-1":"B"}
Explanation
Emphysema is a lung disease that involves damage to the alveoli (tiny air sacs in the lungs), causing them to become stretched out and lose their elasticity. This leads to an abnormally enlarged gas exchange system and the destruction of alveolar walls
Transudative effusion: An effusion is an abnormal buildup of fluid in a body cavity, such as the pleural cavity around the lungs. Transudative effusions occur when fluid leaks out of blood vessels due to changes in pressure or protein levels, rather than from inflammation or injury.
Exudate effusion: This type of effusion occurs when fluid leaks out of blood vessels due to inflammation or injury. The fluid contains high levels of protein and cellular debris, and may be caused by conditions such as pneumonia, cancer, or autoimmune disorders.
D. Abscess: An abscess is a localized collection of pus, usually caused by a bacterial infection. It can occur in various parts of the body, including the lungs.
An abscess in the lung can cause symptoms such as coughing, chest pain, and fever.
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Correct Answer is C
Explanation
Hypertension, or high blood pressure, is a major risk factor for the development of left ventricular hypertrophy (LVH), which is characterized by thickening of the myocardium (heart muscle). LVH occurs in response to increased afterload (the resistance the heart has to pump against), which is a common feature of hypertension.
Angiotensin II, a hormone that is activated in response to hypertension, is a major mediator of LVH in this setting. Angiotensin II causes vasoconstriction (narrowing of blood vessels) and promotes the retention of salt and water, which increases blood volume and pressure. Additionally, angiotensin II stimulates the proliferation of cardiac myocytes (heart muscle cells) and the synthesis of extracellular matrix proteins, leading to hypertrophy and fibrosis of the myocardium.
Other factors that may contribute to LVH in hypertension include increased sympathetic nervous system activity, oxidative stress, and inflammation. However, angiotensin II is considered a key mediator of this process, and drugs that block the renin-angiotensin-aldosterone system (RAAS), such as angiotensinconverting enzyme (ACE) inhibitors and angiotensin receptor blockers (ARBs), are often used to treat hypertension and reduce the risk of LVH and other cardiovascular complications.
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