Which of the following is a primary goal of catheter ablation in the treatment of atrial fibrillation?
To strengthen the heart muscle
To decrease the frequency of ventricular contractions
To permanently eliminate the need for anticoagulation therapy
To restore and maintain normal sinus rhythm
The Correct Answer is D
Rationale:
A. Catheter ablation does not directly improve myocardial contractility; its purpose is focused on electrical conduction rather than muscle strength.
B. While controlling ventricular response may occur with medications, ablation targets the atrial electrical pathways rather than directly controlling ventricular rate.
C. Anticoagulation decisions depend on stroke risk (e.g., CHA₂DS₂-VASc score); ablation does not automatically remove the need for anticoagulants.
D. The primary goal of catheter ablation in atrial fibrillation is to eliminate abnormal electrical pathways causing arrhythmia, thereby restoring and maintaining normal sinus rhythm and reducing symptoms.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Rationale:
A. A normal QRS duration is 0.06–0.10 seconds. A duration of 0.20 seconds indicates a ventricular conduction delay or bundle branch block, which is not normal sinus rhythm.
B. A normal PR interval is 0.12–0.20 seconds. A measurement of 0.22 seconds suggests a first-degree heart block, which is an abnormal finding.
C. Normally, T waves are upright in most leads. Inverted T waves can indicate ischemia, infarction, or electrolyte disturbances, which are not consistent with normal sinus rhythm.
D. In normal sinus rhythm, the P wave precedes each QRS complex, reflecting proper atrial depolarization followed by ventricular depolarization. This relationship confirms that the electrical impulse is originating from the sinoatrial (SA) node, which defines normal sinus rhythm.
Correct Answer is ["A","C"]
Explanation
Rationale:
A. ESR is a nonspecific marker of inflammation. In pericarditis, inflammation of the pericardium leads to an elevated ESR, reflecting the systemic inflammatory response.
B. BNP is elevated in heart failure due to ventricular stretch and volume overload. While pericarditis can affect cardiac function, BNP is not a primary diagnostic marker for pericarditis unless there is associated heart failure.
C. CRP is another nonspecific marker of inflammation. It is commonly elevated in pericarditis and correlates with disease activity and severity.
D. Blood urea nitrogen is a marker of renal function and is not typically affected by pericarditis unless there is concurrent kidney impairment.
E. TSH is used to assess thyroid function and is not related to the inflammatory process seen in pericarditis.
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