What is the earliest clinical manifestation of biliary atresia?
Vomiting.
Hepatomegaly.
Absence of stooling.
Jaundice.
The Correct Answer is D
The correct answer is Choice D: Jaundice.
Choice A rationale:
Vomiting can occur in biliary atresia due to the blockage of bile flow, but it is not the earliest clinical manifestation. Jaundice tends to precede vomiting and is a more characteristic early sign of biliary atresia.
Choice B rationale:
Hepatomegaly (enlargement of the liver) is a common finding in biliary atresia, but it usually occurs after the onset of jaundice. Jaundice is an earlier and more specific manifestation of this condition.
Choice C rationale:
Absence of stooling is a sign that might occur in biliary atresia due to the obstructed bile flow, but it is not typically the earliest clinical manifestation. Jaundice is generally the first noticeable sign.
Choice D rationale:
Jaundice is the earliest clinical manifestation of biliary atresia. It is caused by the buildup of bilirubin in the blood due to impaired bile flow from the liver. The jaundice in biliary atresia is typically progressive and can be one of the key indicators for further evaluation and diagnosis.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
The correct answer is choice B: Pulmonic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy.
Choice A rationale:
Pulmonic stenosis, ventricular septal defect, aortic hypertrophy, left ventricular hypertrophy. This choice is incorrect because it includes "aortic hypertrophy" and "left ventricular hypertrophy," which are not components of the tetralogy of Fallot. Aortic hypertrophy is not a recognized structural defect in tetralogy of Fallot, and left ventricular hypertrophy is not a characteristic feature of this congenital heart condition.
Choice B rationale:
Pulmonic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy. This is the correct choice. Tetralogy of Fallot is characterized by four specific structural defects: pulmonic stenosis (narrowing of the pulmonary valve), ventricular septal defect (hole between the right and left ventricles), overriding aorta (aorta positioned over the ventricular septal defect, receiving blood from both ventricles), and right ventricular hypertrophy (enlargement of the right ventricle due to increased workload).
Choice C rationale:
Aortic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy. This choice is incorrect because it includes "aortic stenosis," which is not part of the tetralogy of Fallot. In tetralogy of Fallot, the stenosis occurs at the pulmonary valve, not the aortic valve.
Choice D rationale:
Aortic stenosis, atrial septal defect, overriding aorta, left ventricular hypertrophy. This choice is incorrect. While "overriding aorta" is present in tetralogy of Fallot, "atrial septal defect" and "left ventricular hypertrophy" are not part of this condition. Atrial septal defects involve a hole between the two atria, not the ventricles, and left ventricular hypertrophy is not typically seen in tetralogy of Fallot.
Correct Answer is ["C","D"]
Explanation
The correct answers are choices C. Novolin R, and D. NovoLog.
Choice A rationale:
Novolin N is an intermediate-acting insulin, not rapid or short acting. It has a slower onset and longer duration of action, making it unsuitable for rapid blood sugar control.
Choice B rationale:
Lantus is a long-acting insulin that provides basal insulin coverage and has a relatively steady effect over 24 hours. It is not rapid or short acting and is used to provide a baseline level of insulin, not for immediate blood sugar control.
Choice C rationale:
Novolin R, also known as regular insulin, is a short-acting insulin with an onset of about 30 minutes and a peak effect around 2 to 3 hours. It is often used to cover mealtime blood sugar elevations and is suitable for short-term blood sugar control.
Choice D rationale:
NovoLog is a rapid-acting insulin analog with an onset of about 15 minutes and a peak effect within 1 to 2 hours. It is designed to mimic the body's rapid insulin release after meals, making it effective for controlling postprandial blood sugar levels.
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