The nurse is caring for a client experiencing sympathetic nervous system stimulation. What assessment finding supports this diagnosis? Select all that apply. (Select All that Apply.)
Increased heart rate
Decrease in urinary bladder muscle tone
Increased blood pressure
Decreased salivation
Decreased bowel sounds
Correct Answer : A,C
A. Increased heart rate: Sympathetic nervous system stimulation typically leads to increased heart rate as part of the "fight or flight" response.
B. Decrease in urinary bladder muscle tone: Sympathetic stimulation would typically cause relaxation of the urinary bladder, leading to increased muscle tone.
C. Increased blood pressure: Sympathetic nervous system activation results in vasoconstriction and increased cardiac output, leading to elevated blood pressure.
D. Decreased salivation: Sympathetic stimulation can lead to decreased salivation as part of the "fight or flight" response, but it's not a consistent finding.
E. Decreased bowel sounds: Sympathetic activation can inhibit gastrointestinal motility, leading to decreased bowel sounds, but it's not a universal finding in sympathetic stimulation.
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Related Questions
Correct Answer is D
Explanation
A. Tachycardia is not typically associated with cholinergic drug effects. Cholinergic stimulation tends to slow down the heart rate rather than increase it.
B. Palpitations may occur with certain cardiac arrhythmias or in response to sympathetic stimulation, but they are not typically associated with cholinergic drug effects.
C. Cholinergic drugs are more likely to cause vasodilation rather than vasoconstriction.
Therefore, vasoconstriction is not a common cardiovascular effect of cholinergic drug therapy.
D. Bradycardia is a common cardiovascular effect of cholinergic drug therapy. Cholinergic stimulation slows down the heart rate by increasing parasympathetic activity, particularly at the
sinoatrial (SA) node. Therefore, bradycardia is the cardiovascular effect that nurses should monitor for when administering cholinergic drugs.
Correct Answer is B
Explanation
A. Decreased urine output is not the primary intended effect of an adrenergic agonist in the
treatment of shock. While adrenergic agonists may increase blood pressure, leading to a decrease in urine output due to vasoconstriction, the primary goal of administering these drugs in shock is to improve tissue perfusion and cardiac output.
B. Increased cardiac output is the primary intended effect of adrenergic agonists in the treatment of shock. These medications stimulate adrenergic receptors, leading to increased heart rate, contractility, and stroke volume, ultimately improving cardiac output and tissue perfusion.
C. Volume restoration may be a secondary effect of administering fluids along with adrenergic agonists in the treatment of shock, but it is not the primary intended effect of the medication
itself.
D. Reduced anxiety is not a primary goal of administering adrenergic agonists in the treatment of shock. While these medications may have anxiolytic effects in certain situations, the primary goal is to improve cardiovascular function and tissue perfusion.
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