Clients taking metformin (Glucophage) should be monitored for this rare but potentially fatal adverse effect:
respiratory acidosis
hyperlipidemia
hypertension
lactic acidosis
The Correct Answer is D
Respiratory acidosis: This is not a typical side effect of metformin.
B. Hyperlipidaemia: Metformin might not directly cause significant hyperlipidaemia.
C. Hypertension: Metformin doesn't typically cause hypertension.
D. Lactic acidosis: This is a rare but serious complication of metformin therapy. It can occur if metformin accumulates in the body due to impaired kidney function or other factors. Monitoring for signs and symptoms is crucial.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Fluoroquinolones: Fluoroquinolones are a class of antibiotics known to have potential side effects like cartilage damage and tendon rupture, especially in younger individuals.
B. Aminoglycosides: While aminoglycosides can have other side effects, they are not typically associated with cartilage or tendon problems.
C. Penicillins: Penicillins can cause allergic reactions but not cartilage or tendon issues.
D. Sulfonamides: Sulfonamides can have various side effects, but cartilage or tendon problems are not common.
Correct Answer is B
Explanation
A. Ketoacids: This is incorrect. While ketoacids are a by-product of fat metabolism that occurs when insulin is low, they do not directly stimulate insulin secretion.
B. Hyperglycaemia: This is correct. Elevated blood glucose levels (hyperglycaemia) directly stimulate the pancreas to secrete insulin to help lower blood glucose levels.
C. Hypoglycaemia: This is incorrect. Low blood glucose levels (hypoglycaemia) would reduce insulin secretion and might increase glucagon secretion to raise blood glucose levels.
D. Glucagon: This is incorrect in the context of directly stimulating insulin secretion. Glucagon is a hormone secreted by the alpha cells of the pancreas when blood glucose levels are low. Its primary role is to increase blood glucose levels by stimulating glycogenolysis and gluconeogenesis in the liver.
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