A young girl with a history of asthma is brought to her family nurse practitioner (FNP). The child is wheezing and has prolonged expirations. The basic etiology of her signs and symptoms (S&S) includes:
use of accessory muscles for breathing.
bronchial-lining swelling that prevents ease of exhalation.
destruction of elastic recoil properties of the lungs.
difficulty with inhalation due to stiff alveoli that have filled with fluid.
The Correct Answer is B
A. Accessory muscle use is a response to respiratory distress, not the underlying cause of asthma symptoms.
B. Asthma causes inflammation and swelling of the bronchial lining, which narrows the airways and makes it difficult to exhale air from the lungs, leading to prolonged expiration and wheezing.
C. Destruction of elastic recoil is associated with chronic lung conditions like emphysema, not asthma.
D. Stiff alveoli and fluid-filled lungs are more characteristic of restrictive lung diseases or pulmonary edema, not asthma.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Troponin is a biomarker used to diagnose myocardial infarction (heart attack), not heart failure.
B. Calcium levels are important for overall metabolic function but are not specific for heart failure.
C. B-type natriuretic peptide (BNP) is elevated in heart failure, specifically in left heart failure, and helps confirm the diagnosis. BNP is a hormone released by the ventricles in response to increased pressure and volume.
D. C-reactive protein (CRP) is a marker of inflammation but does not specifically confirm a heart failure diagnosis.
Correct Answer is D
Explanation
A. Chancres are not associated with glomerulonephritis; this option is incorrect as it refers to syphilis, which does not cause this condition.
B. Glomerulonephritis typically leads to decreased urine output due to reduced GFR, not increased urine output.
C. Poor skin turgor is usually indicative of dehydration rather than hyperproteinemia; in glomerulonephritis, the issue is typically a loss of proteins in the urine, leading to edema.
D. In post-streptococcal glomerulonephritis, hypoproteinemia (loss of protein due to kidney damage) causes fluid to shift from the vascular space to tissues, resulting in edema.
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