A patient with alcoholic liver disease has severe anemia. Which of the following explains the development of anemia in this particular patient?
Alcohol suppresses erythropoiesis.
Alcoholics are often deficient in folate.
Liver dysfunction leads to decreased clotting factors.
Gastric ulcers may lead to chronic blood loss.
Alcohol causes inflammation, which leads to anemia.
The Correct Answer is A
A. Alcohol can directly suppress the production of red blood cells (erythropoiesis) in the bone marrow, leading to anemia.
B. While alcoholics may indeed be deficient in folate due to poor nutrition, folate deficiency is not the primary mechanism for anemia in alcoholic liver disease.
C. Liver dysfunction can lead to decreased production of clotting factors, contributing to coagulopathy, but it is not the primary mechanism for anemia in alcoholic liver disease.
D. Gastric ulcers may indeed lead to chronic blood loss, contributing to anemia, but it is not specific to alcoholic liver disease.
E. While alcohol-induced inflammation may contribute to various complications, including liver damage, it is not the primary mechanism for anemia in alcoholic liver disease.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Abnormal vaginal bleeding. Abnormal vaginal bleeding, especially postmenopausal bleeding, is the most common symptom reported by clients being evaluated for endometrial cancer.
B. Recurrent urinary tract infections. This is not a common symptom of endometrial cancer.
C. Hot flashes. Hot flashes are typically associated with menopause and hormone changes, not endometrial cancer.
D. Blood in the stool. Blood in the stool is more commonly associated with gastrointestinal issues or colorectal cancer, not endometrial cancer.
Correct Answer is ["C","D"]
Explanation
A. The patient experiences a decrease in hemoglobin S. Hydroxyurea does not decrease hemoglobin S levels directly; it works by increasing fetal hemoglobin (Hgb F) levels.
B. The patient experiences dehydration due to diuresis. This is not an indication that Hydroxyurea is working; it is a potential side effect that should be monitored.
C. The patient experiences an increase in fetal hemoglobin (Hbg F). Hydroxyurea works by increasing the levels of fetal hemoglobin, which reduces the sickling of red blood cells.
D. The patient needs fewer blood transfusions. Successful treatment with Hydroxyurea should reduce the frequency of vaso-occlusive crises and the need for blood transfusions.
E. The patient experiences diuresis. This is not an indicator of the medication's effectiveness; it is a potential side effect.
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