A patient with alcoholic liver disease has severe anemia. Which of the following explains the development of anemia in this particular patient?
Alcohol suppresses erythropoiesis.
Alcoholics are often deficient in folate.
Liver dysfunction leads to decreased clotting factors.
Gastric ulcers may lead to chronic blood loss.
Alcohol causes inflammation, which leads to anemia.
The Correct Answer is A
A. Alcohol can directly suppress the production of red blood cells (erythropoiesis) in the bone marrow, leading to anemia.
B. While alcoholics may indeed be deficient in folate due to poor nutrition, folate deficiency is not the primary mechanism for anemia in alcoholic liver disease.
C. Liver dysfunction can lead to decreased production of clotting factors, contributing to coagulopathy, but it is not the primary mechanism for anemia in alcoholic liver disease.
D. Gastric ulcers may indeed lead to chronic blood loss, contributing to anemia, but it is not specific to alcoholic liver disease.
E. While alcohol-induced inflammation may contribute to various complications, including liver damage, it is not the primary mechanism for anemia in alcoholic liver disease.
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Related Questions
Correct Answer is D
Explanation
A. Skin color: While changes in skin color can occur in hemochromatosis due to excess iron deposition, it is not the most important parameter to monitor for the effectiveness of deferoxamine.
B. Liver function: Liver function tests can be abnormal in hemochromatosis due to iron overload, but monitoring liver function alone may not adequately assess the effectiveness of deferoxamine in reducing iron levels.
C. Hematocrit: Hematocrit measures the proportion of blood that is made up of red blood cells and may be elevated in hemochromatosis, but it is not the primary parameter to monitor the effectiveness of deferoxamine.
D. Serum iron level: Serum iron level is the most direct indicator of iron overload and the effectiveness of deferoxamine in chelating and removing excess iron from the body.
Correct Answer is A
Explanation
A. Prolonged bleeding or easy bruising is a characteristic feature of hemophilia, a genetic
disorder characterized by deficient or defective blood clotting proteins, leading to impaired blood clotting and prolonged bleeding after injury or trauma.
B. Excessive blood clotting is not a characteristic feature of hemophilia; rather, it is the opposite.
Hemophilia is characterized by a deficiency in clotting factors, which results in impaired clot formation.
C. Hemophilia does not typically result in increased platelet count. Platelets are involved in the initial stages of blood clotting, but hemophilia primarily affects clotting factor proteins.
D. Delayed wound healing is not a characteristic feature of hemophilia. Hemophilia primarily affects blood clotting, leading to prolonged bleeding, but it does not directly affect wound
healing processes.
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