A nurse is assessing a client who has hypoxemic respiratory failure. Which of the following findings should the nurse expect?
Pulmonary edema
Opioid toxicity
Myasthenia gravis
Flail chest
The Correct Answer is A
A. Pulmonary edema: Pulmonary edema occurs when fluid accumulates in the lungs, impairing gas exchange and leading to hypoxemia. This condition is a common cause of hypoxemic respiratory failure, and patients often present with symptoms such as dyspnea, crackles on auscultation, and hypoxemia. The nurse would expect this finding in a client experiencing hypoxemic respiratory failure.
B. Opioid toxicity: Opioid toxicity can cause respiratory depression, but it typically leads to hypoventilation, which results in hypercapnic respiratory failure (elevated carbon dioxide levels) rather than hypoxemic respiratory failure. Therefore, while opioid toxicity affects breathing, it is not directly associated with hypoxemic respiratory failure.
C. Myasthenia gravis: Myasthenia gravis is a neuromuscular disorder that leads to muscle weakness, including respiratory muscles. While it can cause respiratory failure in severe cases, it does not directly cause hypoxemic respiratory failure. Respiratory failure in myasthenia gravis is often due to insufficient ventilation rather than impaired oxygenation, so this is not the expected finding in hypoxemic respiratory failure.
D. Flail chest: Flail chest occurs due to multiple rib fractures that result in a segment of the chest wall moving paradoxically, impairing ventilation. While this condition can lead to respiratory failure, it typically causes hypercapnic rather than hypoxemic respiratory failure. Therefore, it is less likely to be the cause of hypoxemic respiratory failure in this scenario.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. "Damage to the brain is related to coup and contrecoup injuries. Coup and contrecoup injuries are associated with closed traumatic brain injuries, where the brain hits the skull from forceful movement, such as in a car accident. These do not apply to penetrating injuries, where an object physically enters the brain tissue.
B. "Damage to the brain is related to the size, route, and rate of speed of the object entering the brain." In penetrating traumatic brain injuries, the extent of damage depends largely on the characteristics of the penetrating object—how large it is, the path it takes through the brain tissue, and how fast it enters. These factors determine the depth, location, and severity of brain tissue destruction.
C. "Damage occurs from the penetrating object shattering the skull and causing an infection."
While skull fracture and subsequent infection are possible complications, they are not the primary mechanisms of damage in penetrating brain injuries. The direct trauma caused by the object itself as it disrupts neural tissue is the main source of injury.
D. "Damage occurs from the penetrating injury causing leakage of cerebrospinal fluid." Cerebrospinal fluid (CSF) leakage may occur as a result of a skull or dura mater breach, but this is more of a secondary complication rather than the central mechanism of brain injury. The main damage is from direct tissue penetration and destruction.
Correct Answer is A
Explanation
A. Ataxia: Ataxia is a neurologic manifestation that may occur in diabetes insipidus due to severe dehydration and resulting hypernatremia. Elevated sodium levels can disrupt normal brain function, leading to symptoms like unsteadiness, confusion, muscle twitching, and poor coordination, which may progress if not corrected.
B. Poor skin turgor: Poor skin turgor is a clinical sign of dehydration but reflects fluid loss in the integumentary system rather than a neurologic complication. It helps assess fluid volume status but does not involve cognitive or motor function changes.
C. Hypotension: Hypotension is commonly seen in diabetes insipidus due to volume depletion from excessive urination. However, it is a circulatory effect and does not directly reflect neurologic involvement or function.
D. Dilute urine: Producing large amounts of dilute urine is a defining feature of DI, caused by a deficiency of antidiuretic hormone or kidney resistance to it. This finding pertains to renal output rather than a neurologic complication.
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