A nurse is planning care for a client who is in the manic phase of bipolar disorder.

Which of the following interventions should the nurse include in the client's plan of care?.

Choice A rationale:

Progressive muscle relaxation (PMR) is a well-established technique for stress reduction. It involves systematically tensing and releasing different muscle groups throughout the body. This process helps to release physical tension, promote relaxation, and decrease feelings of stress and anxiety.

PMR has been shown to be effective in reducing stress and anxiety in various populations, including individuals with chronic pain, anxiety disorders, and sleep difficulties.

It is a simple, safe, and accessible technique that can be practiced independently without any special equipment or training.

Choice B rationale:

While sleeping in can provide temporary relief from stress, it is not considered an adaptive coping strategy in the long term.

Oversleeping can disrupt sleep patterns and circadian rhythms, which can actually worsen stress and anxiety in the long run.

It can also lead to social isolation and decreased productivity, further compounding stress.

Choice C rationale:

While distraction techniques, such as thinking about a favorite vacation, can provide temporary relief from stress, they do not address the underlying causes of stress.

Overreliance on distraction can prevent individuals from developing more effective coping strategies for managing stress in the long term.

Choice D rationale:

Social isolation is generally considered a maladaptive coping strategy. While it may provide temporary relief from overwhelming situations, it can lead to increased feelings of loneliness, disconnection, and rumination.

It can also prevent individuals from seeking support and developing more effective coping strategies.

Choice E rationale:

Social support is a crucial factor in stress management. Connecting with supportive friends and loved ones can provide emotional comfort, reduce feelings of isolation, and promote a sense of belonging.

Positive social interactions can also buffer the negative effects of stress and promote resilience.

Choice A rationale:

Seizures are a potentially serious complication of alcohol withdrawal. They occur due to the sudden decrease in GABA (gamma-aminobutyric acid), a neurotransmitter that has inhibitory effects on the central nervous system. Chronic alcohol consumption leads to an upregulation of GABA receptors in the brain to compensate for the depressant effects of alcohol. When alcohol is abruptly withdrawn, the brain is left in a hyperexcitable state, as there is less GABA to suppress neuronal activity. This hyperexcitability can manifest as seizures.

Mechanism of seizures in alcohol withdrawal:

Neuroadaptation: Chronic alcohol exposure leads to adaptations in neuronal excitability to counteract the depressant effects of alcohol. These adaptations include:

Upregulation of GABA receptors: The brain increases the number of GABA receptors to enhance the inhibitory effects of GABA.

Downregulation of glutamate receptors: The brain decreases the number of glutamate receptors to reduce the excitatory effects of glutamate.

GABA rebound: When alcohol is abruptly withdrawn, the upregulated GABA receptors become hypersensitive, leading to an excessive inhibitory response. This is known as GABA rebound.

Glutamate rebound: Concurrently, the downregulated glutamate receptors become more sensitive, leading to an enhanced excitatory response. This is known as glutamate rebound.

Imbalance: The combination of GABA rebound (excessive inhibition) and glutamate rebound (excessive excitation) creates a state of neuronal hyperexcitability, which can trigger seizures.

Risk factors for seizures in alcohol withdrawal:

Severity of alcohol dependence: Individuals with a long history of heavy alcohol consumption are at higher risk.

Previous alcohol withdrawal seizures: A history of seizures during past withdrawal episodes increases the likelihood of recurrence.

Comorbid medical conditions: Certain medical conditions, such as electrolyte imbalances, head injuries, or infections, can increase the risk of seizures.

Concurrent medication use: Certain medications, such as benzodiazepines or barbiturates, can lower the seizure threshold.

Management of seizures in alcohol withdrawal:

Benzodiazepines: Benzodiazepines are the mainstay of treatment for alcohol withdrawal seizures. They enhance the effects of GABA, helping to suppress neuronal excitability and prevent seizures.

Anticonvulsants: In some cases, anticonvulsants, such as gabapentin or valproate, may be used in addition to benzodiazepines.

Electrolyte replacement: Electrolyte imbalances, such as hyponatremia or hypomagnesemia, can contribute to seizures and should be corrected.

Monitoring: Close monitoring of vital signs, neurological status, and seizure activity is essential to ensure prompt intervention if seizures occur.

Choice B rationale:

Nystagmus, or involuntary eye movements, is not a common symptom of alcohol withdrawal. While it can occur in some cases, it is not considered a primary feature of the syndrome.

Choice C rationale:

Tremors are a very common symptom of alcohol withdrawal. They are caused by the same underlying mechanism as seizures, namely, the hyperexcitability of the central nervous system due to decreased GABA activity. Tremors typically manifest as shaking hands, arms, or legs, and can range in severity from mild to severe.

Choice D rationale:

Hallucinations, both auditory and visual, can occur in alcohol withdrawal. They are thought to be due to a combination of factors, including the hyperexcitability of the central nervous system, disruptions in neurotransmitter systems, and sleep deprivation. Hallucinations can be very distressing and can lead to agitation, confusion, and disorientation.