A nurse assesses a patient’s peripheral IV site and notices edema and tenderness above the site. What action will the nurse take next?

A. Smoking causes you to cough frequently, and the wound might get infected by sputum.

While smoking can indeed contribute to respiratory issues like coughing, linking this directly to wound infection by sputum is not the most accurate explanation of how smoking affects wound healing. The primary concern with smoking and wound healing lies in its effects on circulation and tissue oxygenation rather than the risk of infection due to coughing.

B. Nicotine causes vasoconstriction, so your wound might not get enough blood flow to heal.

This is the best response among the options provided. Nicotine, a major component of cigarette smoke, is known to constrict blood vessels (vasoconstriction). This constriction reduces blood flow to the wound site, leading to decreased delivery of oxygen and nutrients necessary for proper wound healing. It addresses the direct physiological impact of smoking on wound healing and provides a clear explanation for the client.

C. Nicotine causes tar to build up in the wound, and it will impair healing.

While nicotine and other components of tobacco smoke can have detrimental effects on healing, particularly through vasoconstriction, the explanation about tar building up in the wound is not entirely accurate. Tar is more associated with lung damage from smoking rather than direct buildup in external wounds. Therefore, this response is less specific and may confuse the client about the actual mechanism of how smoking affects wound healing.

D. Smoking is bad, and you should stop right away.

While this response emphasizes the importance of smoking cessation, it lacks specificity in explaining how smoking impacts wound healing. Providing specific information about vasoconstriction due to nicotine, as mentioned in option B, would be more helpful in helping the client understand the direct effects of smoking on their chronic wound and why cessation is crucial.

A. Intact skin with nonblanchable redness, painful, warm, soft localized area over a bony prominence

Stage 1 pressure injuries are characterized by intact skin with nonblanchable redness over a localized area, typically over a bony prominence like the sacrum, heel, or elbow. The skin may feel painful, warm, and soft to the touch. Nonblanchable redness means that when pressure is applied to the area, the redness does not fade or blanch (turn white). This stage indicates that tissue damage has occurred, but the skin is still intact.

B. Shallow, open, shiny, dry injury, pink-red wound bed without sloughing or bruising: This description is more indicative of a Stage 2 pressure injury, which involves partial-thickness skin loss with an intact or ruptured blister. The wound bed is usually pink or red, and there is no sloughing or bruising.

C. Full-thickness tissue loss, slough and black eschar in wound bed with undermining and tunneling: This description corresponds to a Stage 3 or Stage 4 pressure injury. Stage 3 involves full-thickness tissue loss with visible subcutaneous fat but no bone, tendon, or muscle exposed. Stage 4 involves extensive tissue loss with exposure of bone, tendon, or muscle. Both stages may include slough (yellow or white tissue) and black eschar (hard, necrotic tissue), along with undermining (tissue destruction under intact skin edges) and tunneling (narrow passageways extending from the wound).

D. Full-thickness tissue loss, subcutaneous fat visible, possible undermining and tunneling: This description also corresponds to a Stage 3 pressure injury, as it involves full-thickness tissue loss with visible subcutaneous fat. The mention of possible undermining and tunneling further suggests a Stage 3 pressure injury.