A client with a history of myocardial infarction (MI) develops pulmonary edema. Which pathophysiological process is likely occurring in this client, leading to fluid accumulation in the lungs?
Increased myocardial contractility
Reduced afterload on the heart
Left ventricular remodeling
Enhanced pulmonary vasodilation
The Correct Answer is C
A) Incorrect. Increased myocardial contractility would not typically lead to left ventricular remodeling but might improve cardiac function.
B) Incorrect. Reduced afterload on the heart may be beneficial, but it is not a primary factor in left ventricular remodeling.
C) Correct. Left ventricular remodeling, often occurring after a myocardial infarction, involves changes in the size and shape of the ventricle, which can impair cardiac function and lead to pulmonary edema.
D) Incorrect. Enhanced pulmonary vasodilation is not a primary cause of left ventricular remodeling or pulmonary edema.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A) Incorrect. The primary role of the lymphatic system in the lungs is not gas exchange but rather fluid removal.
B) Incorrect. While the lymphatic system plays a role in overall fluid balance, it is not primarily responsible for regulating blood pressure.
C) Correct. The lymphatic system helps remove excess interstitial fluid from the lungs, preventing fluid accumulation and contributing to fluid balance.
D) Incorrect. Surfactant production is primarily related to maintaining lung compliance and preventing alveolar collapse but is not a primary function of the lymphatic system.
Correct Answer is C
Explanation
A) Incorrect. Sudden onset of severe chest pain is not a typical finding in chronic pulmonary edema.
B) Incorrect. Profuse hemoptysis is not a common symptom of chronic pulmonary edema.
C) Correct. Bilateral pitting edema in the lower extremities is a common sign of fluid retention in chronic pulmonary edema.
D) Incorrect. While an increased heart rate may be present, palpitations are not a typical finding in chronic pulmonary edema.
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