Which neurotransmitter is associated with the hallucinations, delusions, and bizarre behavior seen in schizophrenia?
Serotonin
Gamma-aminobutyric acid (GABA)
Dopamine
Acetylcholine
The Correct Answer is C
Choice A reason: Serotonin modulates mood and anxiety but is not primarily linked to schizophrenia’s core symptoms. While serotonin imbalances contribute to depression, schizophrenia’s hallucinations and delusions stem from dopamine hyperactivity in the mesolimbic pathway, making serotonin an incorrect choice for this disorder’s pathophysiology.
Choice B reason: GABA inhibits neural activity, and its dysfunction is linked to anxiety or seizures, not schizophrenia’s positive symptoms. Schizophrenia involves dopamine excess in the mesolimbic pathway, not GABA deficits. GABA’s role is secondary, making it an inaccurate choice for explaining hallucinations and delusions.
Choice C reason: Dopamine hyperactivity in the mesolimbic pathway causes hallucinations, delusions, and bizarre behavior in schizophrenia. Excess dopamine signaling disrupts cognitive and perceptual processes, leading to positive symptoms. Antipsychotics target D2 receptors to reduce these effects, confirming dopamine’s central role in schizophrenia’s pathophysiology.
Choice D reason: Acetylcholine is involved in memory and attention, not schizophrenia’s core symptoms. While cholinergic deficits occur in dementia, schizophrenia’s hallucinations and delusions are driven by dopamine dysregulation, not acetylcholine. This makes acetylcholine an incorrect choice for the neurotransmitter associated with these symptoms.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason: Flooding involves intense, immediate exposure to the feared stimulus, like elevators, overwhelming amygdala-driven fear responses. This contrasts with gradual exposure, which calms norepinephrine surges incrementally. Flooding is less controlled and riskier, making it incorrect for the described gradual relaxation technique.
Choice B reason: Systematic desensitization pairs gradual exposure to a fear, like elevators, with relaxation to reduce amygdala hyperactivity and norepinephrine-driven panic. This counterconditions fear responses, promoting calm neural pathways, aligning with the described technique and making it the correct choice for this therapeutic approach.
Choice C reason: Combination therapy involves multiple modalities, like medication and psychotherapy, not specifically gradual exposure with relaxation. The described technique targets phobia-specific amygdala responses, not a broad approach, making combination therapy too vague and incorrect for this targeted intervention.
Choice D reason: Cognitive restructuring modifies thought patterns, not exposure-based fear responses. While it addresses distorted beliefs, the described technique uses relaxation with gradual exposure to reduce norepinephrine-driven panic, not cognitive reframing, making this incorrect for the specific therapeutic method described.
Correct Answer is D
Explanation
Choice A reason: Canceling discharge overrides patient autonomy and recovery progress. Stabilized schizophrenia, managed with antipsychotics targeting dopamine, supports discharge with adherence. This action disregards the patient’s rights and neurobiological stabilization, making it an inappropriate advocacy response.
Choice B reason: Notifying security dismisses family concerns and escalates unnecessarily. Schizophrenia management relies on medication adherence, not coercion. This approach ignores patient rights and family education needs, failing to address neurobiological treatment principles, making it incorrect for advocacy.
Choice C reason: Transferring to long-term care assumes ongoing instability, ignoring current stabilization. Antipsychotics correct dopamine imbalances, supporting outpatient management. This undermines patient autonomy and recovery potential, making it an inappropriate advocacy action for a stabilized patient.
Choice D reason: Explaining medication adherence promotes patient autonomy and recovery. Antipsychotics reduce dopamine-driven hallucinations, ensuring symptom control. Educating the family empowers support for adherence, aligning with patient rights and neurobiological treatment principles, making this the correct advocacy response.
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