Which lab results should be monitored for a patient on furosemide?

Creatinine

Platelets

Chloride

RBC

Answer and Explanation

The Correct Answer is A

Choice A reason: Furosemide, a loop diuretic, increases renal workload, risking kidney injury. Creatinine rises (>1.2 mg/dL) with reduced glomerular filtration, a critical marker to monitor for nephrotoxicity or dehydration in heart failure patients.

Choice B reason: Platelets (150,000-450,000/µL) assess clotting, unaffected by furosemide directly. Diuretics alter volume, not hematopoiesis, so monitoring platelets is irrelevant unless bleeding or unrelated conditions complicate the patient’s status.

Choice C reason: Chloride may drop with furosemide, but it’s less critical than creatinine. Hypochloremia affects acid-base balance, yet renal function takes priority, as kidney damage has broader, immediate implications in therapy.

Choice D reason: RBC count reflects anemia, not furosemide’s primary effects. Diuretics cause hemoconcentration if overused, but creatinine better captures renal impact, making red cell monitoring secondary in this context.

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Correct Answer is A

Explanation

Choice A reason: Low-impact exercise, like walking, enhances collateral circulation in PAD, increasing blood flow to ischemic muscles. It reduces claudication by stimulating angiogenesis and improving endothelial function, a cornerstone of evidence-based management to alleviate symptoms effectively.

Choice B reason: Limiting fluid intake addresses edema, not a primary PAD issue, which involves arterial insufficiency, not venous stasis. This strategy lacks scientific support for claudication relief, as hydration status doesn’t directly influence arterial perfusion.

Choice C reason: Tight shoes restrict circulation, worsening PAD by compressing arteries and exacerbating ischemia. Proper footwear is essential, but constriction contradicts vascular physiology, potentially increasing pain and tissue damage rather than improving blood flow.

Choice D reason: Bed rest reduces activity, promoting stasis and deconditioning in PAD, worsening claudication. Immobility decreases muscle pump action and collateral flow, conflicting with evidence that exercise improves symptoms, making this detrimental to recovery.

A. Teach the patient about administration of insulin. B. Give oral hypoglycemic medications before meals. C. Discuss the role of diet in blood glucose control. D. Evaluate the patient’s use of pancreatic enzymes.

Correct Answer is A

Explanation

Choice A reason: CF-related diabetes (CFRD) from pancreatic damage requires insulin, as glucose of 180-250 mg/dL indicates insulin deficiency. Teaching administration addresses this directly, aligning with standard CFRD management for glycemic control.

Choice B reason: Oral hypoglycemics aren’t effective in CFRD, which stems from insulin lack, not resistance. Glucose levels of 180-250 mg/dL need insulin, making this inappropriate for CF’s unique endocrine pathology.

Choice C reason: Diet impacts glucose, but CFRD requires insulin first, not just dietary control. Levels of 180-250 mg/dL exceed dietary management alone, so this is secondary to initiating insulin therapy in CF.

Choice D reason: Pancreatic enzymes aid digestion in CF, not glucose control directly. Evaluating use is routine, but hyperglycemia of 180-250 mg/dL points to CFRD, necessitating insulin over enzyme adjustment.

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Which lab results should be monitored for a patient on furosemide?

Nursing Test Bank

Naxlex Comprehensive Predictor Exams

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