Which information obtained by the nurse assessing a patient admitted with multiple myeloma is most important to report to the health care provider?
Urine sample has Bence-Jones protein.
Patient is complaining of severe back pain.
Serum calcium level is 15 mg/dL.
Patient reports no stool for 5 days.
The Correct Answer is C
A. Presence of Bence-Jones protein in the urine is indicative of multiple myeloma, but hypercalcemia is a more urgent issue to address.
B. Severe back pain is common in multiple myeloma due to bone involvement, but it is not as immediately life-threatening as hypercalcemia.
C. Elevated serum calcium levels (hypercalcemia) are a common complication of multiple myeloma and can lead to various complications, including kidney damage, altered mental status, and cardiac arrhythmias. Therefore, it is crucial to report significantly elevated serum calcium levels promptly to the health care provider for appropriate management.
D. Patient reports no stool for 5 days may indicate constipation, which is important to address but is not as urgent as hypercalcemia in the context of multiple myeloma.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Cystocele: A cystocele occurs when the bladder bulges into the vaginal canal, which can cause symptoms such as dyspareunia, backache, pelvic pressure, urinary tract infections, and urinary urgency.
B. Fibroid: Fibroids are benign tumors of the uterus and can cause pelvic pain and pressure, but they are not typically associated with urinary symptoms.
C. Ovarian cyst: Ovarian cysts can cause pelvic pain and pressure but are not usually associated with urinary symptoms like urinary urgency.
D. Rectocele: A rectocele occurs when the rectum bulges into the vaginal canal, which can cause symptoms like dyspareunia and pelvic pressure, but it is more likely to cause constipation than urinary urgency.
Correct Answer is A
Explanation
A. Alcohol can directly suppress the production of red blood cells (erythropoiesis) in the bone marrow, leading to anemia.
B. While alcoholics may indeed be deficient in folate due to poor nutrition, folate deficiency is not the primary mechanism for anemia in alcoholic liver disease.
C. Liver dysfunction can lead to decreased production of clotting factors, contributing to coagulopathy, but it is not the primary mechanism for anemia in alcoholic liver disease.
D. Gastric ulcers may indeed lead to chronic blood loss, contributing to anemia, but it is not specific to alcoholic liver disease.
E. While alcohol-induced inflammation may contribute to various complications, including liver damage, it is not the primary mechanism for anemia in alcoholic liver disease.
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