When evaluating a patient who is taking orlistat, which is an intended therapeutic effect?
Decreased weight
Increased appetite
Decreased hyperactivity
Increased wakefulness
The Correct Answer is A
A. Decreased weight: Orlistat is a medication used for weight management by inhibiting the absorption of dietary fats. Its intended therapeutic effect is to promote weight loss by reducing the amount of fat absorbed from the diet.
B. Increased appetite: Orlistat does not increase appetite; rather, it may help control appetite by promoting satiety through its mechanism of action.
C. Decreased hyperactivity: Orlistat is not indicated for the treatment of hyperactivity.
D. Increased wakefulness: Orlistat does not affect wakefulness; it primarily acts in the gastrointestinal tract to inhibit fat absorption.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Carbidopa prevents the breakdown of levodopa in the periphery, specifically in the gastrointestinal tract and peripheral tissues, allowing more levodopa to reach the brain and be converted to dopamine. This enhances the effectiveness of levodopa therapy in managing the symptoms of Parkinson's disease.
B. Carbidopa is not the biologic precursor of dopamine. It is a peripheral decarboxylase inhibitor that does not cross the blood-brain barrier.
C. Carbidopa does not directly allow for larger doses of levodopa to be given. However, by
inhibiting the peripheral breakdown of levodopa, it enhances the availability of levodopa to the central nervous system, potentially improving therapeutic efficacy.
D. While levodopa-carbidopa combination therapy may have fewer drug-food interactions compared to levodopa alone, the primary reason for combining these medications is to enhance the effectiveness of levodopa by preventing its peripheral breakdown.
Correct Answer is C
Explanation
A. CT of the brain can proceed regardless of atropine administration, as it does not directly affect the assessment of brain stem reflexes.
B. Electroencephalogram (EEG) can also proceed regardless of atropine administration, as it evaluates brain electrical activity rather than brain stem reflexes.
C. Assessment of brain stem reflexes should be postponed until all atropine is excreted and no
longer exerting its effects. Atropine can mask or alter the pupillary response and other brain stem reflexes, such as gag reflex and cough reflex, which are important indicators of neurological function.
D. Pupil response assessment should be postponed until atropine effects have dissipated, as
atropine can cause pupillary dilation, which may interfere with accurate assessment of pupil size and reactivity
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