What is the primary result of anemia?
Depressed hematopoietic system.
Presence of abnormal hemoglobin.
Decreased oxygen-carrying capacity of blood.
Increased blood viscosity.
The Correct Answer is C
The correct answer is choice C. Decreased oxygen-carrying capacity of blood.
Choice A rationale:
Anemia does not primarily result in a depressed hematopoietic system. In fact, anemia often occurs due to various factors that affect red blood cell production or lifespan. The hematopoietic system can be overactive in response to anemia, attempting to compensate for the reduced oxygen-carrying capacity of the blood.
Choice B rationale:
While some anemias may involve the presence of abnormal hemoglobin (e.g., sickle cell anemia), this is not the primary result of anemia. The primary consequence of anemia is a decreased ability of the blood to carry oxygen to the body's tissues.
Choice C rationale:
The correct choice. Anemia leads to a decreased oxygen-carrying capacity of the blood. Hemoglobin, the protein in red blood cells responsible for carrying oxygen, is reduced in quantity or function in various types of anemia. This results in inadequate oxygen delivery to tissues, potentially causing symptoms such as fatigue, weakness, pallor, and shortness of breath.
Choice D rationale:
Increased blood viscosity is not a primary result of anemia. Anemia tends to reduce blood viscosity because there are fewer red blood cells and less hemoglobin present, which makes the blood more fluid and less viscous. Increased blood viscosity is more commonly associated with conditions like polycythemia, where there is an excess of red blood cells.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
The correct answer is choice B: Pulmonic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy.
Choice A rationale:
Pulmonic stenosis, ventricular septal defect, aortic hypertrophy, left ventricular hypertrophy. This choice is incorrect because it includes "aortic hypertrophy" and "left ventricular hypertrophy," which are not components of the tetralogy of Fallot. Aortic hypertrophy is not a recognized structural defect in tetralogy of Fallot, and left ventricular hypertrophy is not a characteristic feature of this congenital heart condition.
Choice B rationale:
Pulmonic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy. This is the correct choice. Tetralogy of Fallot is characterized by four specific structural defects: pulmonic stenosis (narrowing of the pulmonary valve), ventricular septal defect (hole between the right and left ventricles), overriding aorta (aorta positioned over the ventricular septal defect, receiving blood from both ventricles), and right ventricular hypertrophy (enlargement of the right ventricle due to increased workload).
Choice C rationale:
Aortic stenosis, ventricular septal defect, overriding aorta, right ventricular hypertrophy. This choice is incorrect because it includes "aortic stenosis," which is not part of the tetralogy of Fallot. In tetralogy of Fallot, the stenosis occurs at the pulmonary valve, not the aortic valve.
Choice D rationale:
Aortic stenosis, atrial septal defect, overriding aorta, left ventricular hypertrophy. This choice is incorrect. While "overriding aorta" is present in tetralogy of Fallot, "atrial septal defect" and "left ventricular hypertrophy" are not part of this condition. Atrial septal defects involve a hole between the two atria, not the ventricles, and left ventricular hypertrophy is not typically seen in tetralogy of Fallot.
Correct Answer is B
Explanation
The correct answer is choice B. First stage.
Choice A rationale:
There is no fourth stage of Lyme disease. Lyme disease typically progresses through three stages: early localized, early disseminated, and late disseminated. The symptoms mentioned in the question are more indicative of earlier stages of the disease.
Choice B rationale:
The child is likely exhibiting symptoms of the first stage of Lyme disease, known as early localized Lyme disease. This stage is characterized by the appearance of small annular (circular) lesions known as erythema migrans. These lesions are often red and have a clear center, resembling a "bull's-eye" pattern. This stage occurs within days to weeks after a tick bite and is usually accompanied by flu-like symptoms.
Choice C rationale:
There is no third stage of Lyme disease. The third stage is considered the late disseminated stage, which occurs months to years after the initial infection. It typically involves more severe symptoms, such as arthritis, neurological issues, and cardiac abnormalities.
Choice D rationale:
There is no second stage of Lyme disease. The second stage is the early disseminated stage, which occurs weeks to a few months after the tick bite. It involves the spread of the bacteria to other parts of the body, leading to symptoms such as multiple erythema migrans lesions, flu-like symptoms, fatigue, and muscle and joint pain.
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