The nurse is performing the preoperative assessment for a client scheduled for a vertebroplasty of the cervical spine. Which finding should the nurse alert the healthcare provider of prior to the procedure?

A. Reduced pain in eczematous areas:
While hydration of the skin may contribute to reduced pain in some cases, the primary goal of urea cream is to moisturize and hydrate the skin rather than directly address pain.

B. Healing with a return to normal skin appearance:
Urea cream can contribute to the healing process by hydrating the skin and promoting the removal of dry, scaly skin. However, complete healing and a return to normal skin appearance may also depend on the underlying cause of eczema and other factors.

C. Decreased weeping of ulcerations in affected areas:
Urea cream can help reduce excessive dryness and weeping in eczematous areas by promoting hydration and moisture balance. However, it may not directly address ulcerations, and other interventions may be needed for open wounds.

D. Hydration of affected dry skin areas:
This is the correct answer. Urea is a natural moisturizing factor that helps retain water in the skin. Applying urea cream to affected dry skin areas is expected to hydrate the skin, reduce dryness, and improve the overall moisture balance.

A. Hyperaldosteronism causing an increased sodium reabsorption in renal tubules.
Hyperaldosteronism is characterized by an excess of aldosterone, a hormone that regulates sodium and water balance. In cirrhosis, however, sodium retention is often related to other mechanisms such as portal hypertension and hypoalbuminemia, rather than hyperaldosteronism.

B. Decreased renin-angiotensin response related to an increase in renal blood flow.
Cirrhosis is more commonly associated with an activated renin-angiotensin-aldosterone system, leading to increased sodium and water retention. The increased renin-angiotensin response is a compensatory mechanism to maintain perfusion in the setting of cirrhosis and does not contribute to decreased renal blood flow.

C. Decreased portacaval pressure with greater collateral circulation.
This statement is not accurate. In cirrhosis, there is typically increased portacaval pressure due to portal hypertension, which can lead to the development of collateral circulation. However, this does not explain the edema and ascites seen in cirrhosis.

D. Hypoalbuminemia that results in a decreased colloidal oncotic pressure.
This is the correct choice. In cirrhosis, liver damage leads to decreased synthesis of albumin. Albumin plays a crucial role in maintaining colloidal oncotic pressure, and when it is decreased (hypoalbuminemia), fluid is more likely to leak out of blood vessels, resulting in edema. The same mechanism contributes to the development of ascites in the abdominal cavity.