The nurse is caring for a client with a history of deep vein thrombosis. Which intervention should the nurse include in the care plan?

Choice A reason: HIV does not primarily cause a deficiency in antibody production. B-cells produce antibodies, but HIV targets CD4 T-cells, impairing their ability to activate B-cells. This indirectly reduces antibody effectiveness, but the primary mechanism is T-cell destruction, not a direct antibody production deficit, making this incorrect.

Choice B reason: HIV infects and destroys helper T-cells (CD4 cells), critical for coordinating immune responses. By reducing CD4 cell counts, HIV impairs activation of B-cells and cytotoxic T-cells, leading to immune suppression. This is the primary mechanism of AIDS-related immune deficiency, making it the correct explanation for HIV pathology.

Choice C reason: Proliferation of suppressor T-cells (regulatory T-cells) is not a primary HIV mechanism. HIV depletes CD4 cells, not suppressor T-cells, which modulate immune responses. While immune dysregulation occurs, the hallmark is CD4 destruction, not suppressor T-cell proliferation, making this an inaccurate description of HIV’s action.

Choice D reason: HIV does not increase B-lymphocyte numbers. It impairs B-cell function indirectly by destroying CD4 cells, which are needed to activate B-cells for antibody production. B-cell hyperactivity may occur in early HIV, but the primary immune suppression results from CD4 cell loss, not B-cell proliferation.

Choice A reason: Hepatorenal failure involves liver and kidney dysfunction, typically from chronic liver disease or shock. Elevated amylase and lipase, with pain and vomiting, point to pancreatic inflammation, not hepatorenal issues. Pancreatitis is more consistent with the client’s post-cholecystectomy presentation and lab findings.

Choice B reason: Biliary duct obstruction causes jaundice and elevated bilirubin, not amylase or lipase. Pain may radiate, but vomiting and fever with high pancreatic enzymes suggest pancreatitis, a known post-cholecystectomy complication, rather than a blocked bile duct, which lacks pancreatic enzyme elevation.

Choice C reason: Surgical site infection causes localized pain, erythema, and fever but not elevated amylase or lipase. Radiating pain and vomiting align with pancreatitis, not wound infection. Pancreatic enzyme elevation is specific to pancreatic inflammation, making infection less likely than pancreatitis in this scenario.

Choice D reason: Acute pancreatitis is characterized by elevated amylase and lipase, epigastric pain radiating to the back, vomiting, and fever. Post-cholecystectomy, bile duct manipulation or gallstone migration can trigger pancreatitis. These findings align perfectly with the client’s symptoms and lab results, making it the correct condition.