Which treatment is likely to be planned for a patient who develops pernicious anemia after a gastrectomy?
Triple-drug therapy.
IV therapy.
Quadruple-drug therapy.
Cobalamin replacement therapy.
The Correct Answer is D
Choice A rationale:
Triple-drug therapy is not the standard treatment for pernicious anemia after a gastrectomy. Pernicious anemia is primarily caused by vitamin B12 deficiency due to the absence of intrinsic factor, which is essential for vitamin B12 absorption.
Choice B rationale:
IV therapy is a broad term and does not specify the treatment for pernicious anemia. In the context of pernicious anemia, cobalamin replacement therapy administered via intramuscular injections is the preferred treatment.
Choice C rationale:
Quadruple-drug therapy is not a recognized treatment for pernicious anemia. The primary treatment for pernicious anemia involves cobalamin replacement therapy to address the vitamin B12 deficiency.
Choice D rationale:
Cobalamin replacement therapy is the appropriate treatment for pernicious anemia after a gastrectomy. Since the patient lacks intrinsic factor, which is necessary for vitamin B12 absorption, cobalamin replacement therapy bypasses the need for intrinsic factor and provides the necessary vitamin B12 directly.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A rationale:
Serotonin is a neurotransmitter that plays a role in mood regulation, sleep, and other functions in the body. It is not a component that forms an adhesive bridge between platelets and vascular subendothelial structures in the clotting process.
Choice B rationale:
Platelet factor III is not a component that forms an adhesive bridge between platelets and vascular subendothelial structures in the clotting process. Platelet factor III is not widely recognized in the context of clotting; it is more commonly referred to as tissue factor and is involved in the extrinsic pathway of coagulation.
Choice C rationale:
Von Willebrand factor (VWF) is a protein that plays a crucial role in hemostasis. It forms an adhesive bridge between platelets and vascular subendothelial structures, particularly at sites of vascular injury. VWF helps platelets adhere to the injured vessel wall, leading to the formation of a stable blood clot. This process is essential for preventing excessive bleeding. A deficiency or dysfunction in VWF can lead to von Willebrand disease, a bleeding disorder characterized by prolonged bleeding times.
Choice D rationale:
Adenosine diphosphate (ADP) is a molecule released by platelets when they are activated. ADP helps in the aggregation of platelets, but it does not directly form an adhesive bridge between platelets and vascular subendothelial structures.
Correct Answer is A
Explanation
Choice A rationale:
Chronic stable angina typically occurs with both activity and rest due to the increased oxygen demand of the heart during exertion. It is relieved with rest or nitroglycerin, which helps to decrease the heart's workload. This choice accurately represents the characteristic of chronic stable angina.
Choice B rationale:
Chronic stable angina usually lasts for a short duration, usually around 5 minutes, and is relieved with rest or nitroglycerin. Prolonged pain lasting 10-15 minutes or more could indicate an unstable angina or myocardial infarction. This choice is incorrect for chronic stable angina.
Choice C rationale:
Pain due to chronic stable angina subsides when the precipitating factor, such as physical exertion, is resolved or with medication like nitroglycerin. Therefore, this statement is incorrect for chronic stable angina.
Choice D rationale:
Chronic stable angina follows a predictable pattern, occurring intermittently over a period of time, and is usually triggered by physical exertion or emotional stress. This option does not accurately represent the typical pattern of chronic stable angina.
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