After a client has been prescribed fluoxetine (Prozac) for a diagnosis of anxiety disorder, which of the following information should the nurse be sure to include in the client teaching?
The client should not stop this medication abruptly to avoid discontinuation syndrome
The client may experience frequent constipation and should increase their intake of dietary fiber
This medication takes 4-6 months to be effective, the client should be told to be patient
This medication can cause addiction, so the client should not take more than prescribed
The Correct Answer is A
Choice A reason: Abruptly stopping fluoxetine, an SSRI, disrupts serotonin levels, causing discontinuation syndrome with symptoms like dizziness and irritability due to rapid neurotransmitter imbalance in the brain. Gradual tapering stabilizes serotonin, preventing withdrawal, making this critical teaching for safe medication management in anxiety treatment.
Choice B reason: Constipation is not a common side effect of fluoxetine, which primarily causes nausea or diarrhea via serotonin modulation. Anticholinergic drugs, not SSRIs, typically cause constipation. This teaching is inaccurate, as fluoxetine’s side effect profile does not emphasize gastrointestinal slowing, making it incorrect.
Choice C reason: Fluoxetine takes 4-8 weeks, not months, to reach efficacy by increasing serotonin in the prefrontal cortex and amygdala. Overstating the timeline discourages adherence, as patients expect faster relief from anxiety symptoms, making this teaching point scientifically inaccurate and misleading.
Choice D reason: Fluoxetine is not addictive, as it lacks the reinforcing GABA effects of benzodiazepines. It modulates serotonin for anxiety without dependence risk. This teaching is incorrect, as it misrepresents fluoxetine’s pharmacological profile, potentially causing unnecessary fear about its safe use in treatment.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Asking about faith in stress assesses coping strategies, as faith can modulate stress responses via the hypothalamic-pituitary-adrenal axis, reducing cortisol levels. This explores psychological resilience, not just religious beliefs, aligning with holistic assessment of how patients manage stressors impacting mental health.
Choice B reason: Religious affiliation focuses on specific beliefs or denominations, not their role in stress management. The question targets coping, not affiliation details. Faith’s impact on stress involves neurobiological calming effects, making this option too narrow and incorrect for the assessment topic.
Choice C reason: Educational background is unrelated to faith’s role in stress. Coping involves psychological and neurobiological mechanisms, like serotonin modulation, not academic history. The question assesses emotional resilience, not education, making this option irrelevant to the described assessment focus.
Choice D reason: Culture includes broader societal norms, not specifically faith’s role in coping. While faith may be cultural, the question targets stress management, linked to neurobiological stress responses, not cultural identity. Coping strategies is the more precise assessment topic, making culture incorrect.
Correct Answer is A
Explanation
Choice A reason: Abruptly stopping fluoxetine, an SSRI, disrupts serotonin levels, causing discontinuation syndrome with symptoms like dizziness and irritability due to rapid neurotransmitter imbalance in the brain. Gradual tapering stabilizes serotonin, preventing withdrawal, making this critical teaching for safe medication management in anxiety treatment.
Choice B reason: Constipation is not a common side effect of fluoxetine, which primarily causes nausea or diarrhea via serotonin modulation. Anticholinergic drugs, not SSRIs, typically cause constipation. This teaching is inaccurate, as fluoxetine’s side effect profile does not emphasize gastrointestinal slowing, making it incorrect.
Choice C reason: Fluoxetine takes 4-8 weeks, not months, to reach efficacy by increasing serotonin in the prefrontal cortex and amygdala. Overstating the timeline discourages adherence, as patients expect faster relief from anxiety symptoms, making this teaching point scientifically inaccurate and misleading.
Choice D reason: Fluoxetine is not addictive, as it lacks the reinforcing GABA effects of benzodiazepines. It modulates serotonin for anxiety without dependence risk. This teaching is incorrect, as it misrepresents fluoxetine’s pharmacological profile, potentially causing unnecessary fear about its safe use in treatment.
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