A patient, diagnosed with Addison's disease, is admitted to the emergency department (ED) after spending the day at the lake. The patient is diaphoretic, weak and forgetful. Which intervention would be supportive for these S&S (signs and symptoms)?

A. Diminished serum albumin levels cause water to shift from blood to tissue: In cirrhosis, liver dysfunction leads to decreased production of albumin, a protein that helps maintain oncotic pressure. Low albumin levels cause fluid to shift from the vascular space into the tissues, resulting in ascites and peripheral edema.

B. Portal hypotension causes a fluid shift from the abdominal cavity into the portal veins: Portal hypertension, not hypotension, is a common feature of cirrhosis, but it leads to ascites by increasing pressure in the portal venous system, not by shifting fluid into the portal veins.

C. Hypoaldosteronism causes a fluid volume deficit: shifting water from blood into tissue: Cirrhosis often leads to hyperaldosteronism, not hypoaldosteronism, resulting in sodium and water retention, which contributes to edema.

D. Aberrations of the portal system cause a back-up of blood that leads to hydronephrosis: Hydronephrosis is related to obstruction of the urinary tract, not a complication of portal hypertension or cirrhosis.

A. Colon polyps: Colon polyps are not treated with antibiotics or antacids and are unrelated to Helicobacter pylori infection.

B. Atrophic gastritis: Atrophic gastritis involves chronic inflammation and thinning of the stomach lining and is not typically treated with antibiotics for Helicobacter pylori.

C. Intussusception of the small bowel: Intussusception is a condition where part of the intestine telescopes into itself and is not related to Helicobacter pylori.

D. Peptic ulcer disease: Peptic ulcer disease, especially gastric or duodenal ulcers, is commonly associated with Helicobacter pylori infection, which is treated with antibiotics and antacids.