A nurse is providing care for a client who has diabetes insipidus (DI). Which of the following is a cause of acquired central diabetes insipidus?
Hypokalemia
Surgery
Renal failure
Sickle cell disease
The Correct Answer is B
Choice A Reason:
Hypokalemia, or low potassium levels, is not a direct cause of central diabetes insipidus. Central diabetes insipidus is primarily related to issues with the production or release of antidiuretic hormone (ADH) from the hypothalamus or pituitary gland. Hypokalemia can affect kidney function but does not typically cause central diabetes insipidus.
Choice B Reason:
Surgery, particularly brain surgery, can cause central diabetes insipidus by damaging the hypothalamus or pituitary gland. These structures are crucial for the production and release of ADH, which regulates water balance in the body. Damage to these areas during surgery can lead to a deficiency in ADH, resulting in central diabetes insipidus.
Choice C Reason:
Renal failure is not a cause of central diabetes insipidus. While renal failure affects the kidneys’ ability to filter waste and balance fluids, central diabetes insipidus is related to a deficiency in ADH production or release. Renal failure can lead to other types of diabetes insipidus, such as nephrogenic diabetes insipidus, where the kidneys do not respond properly to ADH.
Choice D Reason:
Sickle cell disease is not a direct cause of central diabetes insipidus. Sickle cell disease primarily affects red blood cells and can lead to various complications, including kidney damage. However, it does not typically cause central diabetes insipidus, which is related to issues with ADH production or release.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A Reason:
Conivaptan hydrochloride is a vasopressin receptor antagonist used to treat hyponatremia associated with SIADH. It works by blocking the action of ADH, thereby promoting water excretion without losing sodium. This medication is appropriate for managing SIADH.
Choice B Reason:
Vasopressin, also known as antidiuretic hormone (ADH), is not appropriate for a patient with SIADH. SIADH is characterized by excessive release of ADH, leading to water retention and hyponatremia. Administering vasopressin would exacerbate the condition by increasing water retention and further lowering sodium levels.
Choice C Reason:
Sodium chloride tablets are used to manage hyponatremia by increasing sodium levels in the blood. This treatment is appropriate for patients with SIADH to help correct the sodium imbalance caused by excessive ADH.
Choice D Reason:
Tolvaptan is another vasopressin receptor antagonist that is used to treat hyponatremia associated with SIADH. It helps to increase serum sodium levels by promoting water excretion while retaining sodium. This medication is suitable for managing SIADH.
Correct Answer is A
Explanation
The correct answer is a) Serum potassium levels.
Choice A reason:
Serum potassium levels are crucial to monitor in clients with hypervolemia, ketoacidosis, and metabolic acidosis. Potassium imbalances are common in these conditions due to shifts between intracellular and extracellular compartments. In diabetic ketoacidosis (DKA), for example, insulin deficiency and acidosis cause potassium to move out of cells, leading to hyperkalemia. However, once treatment with insulin begins, potassium shifts back into cells, which can cause hypokalemia. Both hyperkalemia and hypokalemia can have serious cardiac implications, making it essential to monitor and manage potassium levels closely.
Choice B reason:
Serum calcium levels are important for overall health, but they are not the primary concern in the context of hypervolemia, ketoacidosis, and metabolic acidosis. While calcium imbalances can occur, they are less common and less immediately life-threatening compared to potassium imbalances. Monitoring calcium is still necessary, but it does not require the same level of immediate intervention.
Choice C reason:
Serum sodium levels are also important to monitor, especially in conditions like hypervolemia where fluid balance is disrupted. However, sodium imbalances are typically managed through fluid management and do not pose the same immediate risk as potassium imbalances in the context of ketoacidosis and metabolic acidosis. Hyponatremia or hypernatremia can cause neurological symptoms, but these are generally less acute compared to the cardiac risks associated with potassium imbalances.
Choice D reason:
Blood urea nitrogen (BUN) levels provide information about kidney function and hydration status. Elevated BUN can indicate dehydration or renal impairment, which are relevant in the context of hypervolemia and ketoacidosis. However, BUN levels do not require the same level of immediate intervention as potassium levels. Monitoring BUN is important for overall management but is not the primary concern in acute settings.
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