A nurse is planning care immediately following birth for a newborn who has a myelomeningocele that is leaking cerebrospinal fluid. Which of the following actions should the nurse include in the plan of care?

Uterine atony is a condition in which the uterus does not contract properly after delivery, leading to excessive bleeding or postpartum hemorrhage. Uterine atony can be caused by various factors, such as

prolonged or fast labor, multiple gestation, large baby, polyhydramnios, infection, or use of certain medications¹.
The nurse should monitor the client for any signs of uterine atony, such as:
- A soft or boggy uterus that does not respond to massage
- Excessive bleeding or clots from the vagina
- Low blood pressure or fast pulse
- Pale or clammy skin
- Dizziness or fainting
The nurse should also provide immediate interventions to stop the bleeding and restore uterine tone, such
as:
- Massaging the uterus firmly until it contracts
- Administering uterotonic medications as ordered to stimulate uterine contractions
- Administering intravenous fluids and blood products as needed to replace blood loss
- Notifying the provider and preparing for possible surgical procedures if bleeding persists
Uterotonic medications are drugs that cause the uterus to contract and reduce bleeding. They are used to prevent or treat postpartum hemorrhage due to uterine atony. The most common uterotonic medications are:
- Oxytocin: a hormone that is naturally produced by the body during labor and breastfeeding. It is the first- line drug for uterine atony and is given intravenously or intramuscularly. It causes strong and sustained contractions of the uterus and also reduces blood pressure and pain. It has few side effects, but it can cause water retention, nausea, vomiting, or headache in high doses²³.
- Methylergonovine: a synthetic derivative of ergot, a fungus that grows on rye. It is a second-line drug for uterine atony and is given intramuscularly or orally. It causes prolonged contractions of the uterus and also constricts blood vessels in other parts of the body. It can cause side effects such as hypertension, headache, nausea, vomiting, chest pain, or allergic reactions. It is contraindicated in clients with hypertension, preeclampsia, cardiac disease, or liver disease²³.
- Carboprost: a synthetic form of prostaglandin F2 alpha, a hormone that regulates inflammation and blood clotting. It is a third-line drug for uterine atony and is given intramuscularly. It causes intense contractions of the uterus and also relaxes smooth muscles in other parts of the body. It can cause side effects such as fever, diarrhea, nausea, vomiting, bronchospasm, or allergic reactions. It is contraindicated in clients with asthma, liver disease, or kidney disease²³.
- Misoprostol: a synthetic form of prostaglandin E1, a hormone that protects the stomach lining from ulcers. It is an alternative drug for uterine atony and is given orally, rectally, sublingually, or vaginally. It causes mild to moderate contractions of the uterus and also dilates blood vessels in other parts of the body. It can cause side effects such as fever, chills, shivering, nausea, vomiting, diarrhea, or abdominal pain. It is contraindicated in clients with allergy to prostaglandins²³.
Therefore, the nurse should anticipate the use of methylergonovine for a client who has uterine atony that does not respond to oxytocin administration. The nurse should also monitor the client's blood pressure and vital signs closely and report any adverse reactions to the provider.

The other options are not medications that the nurse should anticipate the use of for uterine atony:
- a) Terbutaline is a medication that belongs to a class of drugs called beta-adrenergic agonists. It is used to relax the smooth muscles of the bronchi and uterus. It is used to treat asthma and preterm labor by preventing or stopping contractions. It is not indicated for uterine atony and can cause side effects such as tachycardia, palpitations, tremors, anxiety or hypotension²⁴.
- c) Hydralazine is a medication that belongs to a class of drugs called vasodilators. It is used to lower blood pressure by relaxing the smooth muscles of the arteries. It is used to treat hypertension and preeclampsia by reducing vascular resistance and improving blood flow. It is not indicated

This is the action that the nurse should take after recognizing an early deceleration of the fetal heart rate tracing. Early decelerations are symmetrical decreases and return-to-normal linked to uterine contractions¹. The decrease in heart rate occurs gradually, and the nadir of the deceleration occurs at the same time as the peak of the uterine contraction³. Early decelerations are caused by compression of the fetus's head during a uterine contraction, which can stimulate the vagus nerve and cause a decrease in the fetal heart rate⁴. Early decelerations are nothing to be alarmed about¹. They are considered normal and benign, as they do not affect fetal oxygenation or well-being³. Therefore, the nurse should continue to monitor the client and the fetal heart rate tracing and document the findings.

The other options are not correct because they are not appropriate actions for early decelerations.
a) Assist the client to lay on her right side.
This is not an appropriate action for early decelerations, as they are not caused by maternal position or uteroplacental insufficiency. Changing the maternal position may help improve fetal oxygenation and blood flow in cases of late or variable decelerations, which are signs of fetal distress¹. However, early decelerations do not indicate fetal distress and do not require any intervention.
c) Discontinue the oxytocin.
This is not an appropriate action for early decelerations, as they are not caused by oxytocin administration or uterine hyperstimulation. Oxytocin is a hormone that stimulates uterine contractions and can be used to induce or augment labor. However, excessive or prolonged use of oxytocin can cause uterine fatigue and reduce its ability to contract after delivery, leading to uterine atony and postpartum hemorrhage². Oxytocin can also cause late or variable decelerations, which are signs of fetal distress¹. However, early decelerations do not indicate fetal distress and do not require any intervention.

d) Administer oxygen at 8 L/min per mask.
This is not an appropriate action for early decelerations, as they are not caused by fetal hypoxia or acidosis. Oxygen administration may help improve fetal oxygenation and blood flow in cases of late or variable decelerations, which are signs of fetal distress¹. However, early decelerations do not indicate fetal distress and do not require any intervention.