A client with end-stage kidney disease is scheduled to begin hemodialysis. The nurse is working with the client to adapt the client’s diet to maximize the therapeutic effect and minimize the risks of complications. The client’s diet should include which of the following modifications?

Choice A reason: In the oliguric phase of AKI, kidney function is severely impaired, reducing potassium excretion. This leads to hyperkalemia, which disrupts cardiac electrical activity, potentially causing life-threatening arrhythmias or cardiac arrest. Elevated potassium levels are a hallmark of this phase due to decreased glomerular filtration rate and impaired tubular secretion.

Choice B reason: Urine output of 2000 mL in 24 hours indicates polyuria, characteristic of the recovery phase of AKI, not the oliguric phase, where output is typically less than 400 mL/day. High urine output suggests restored renal function, which is not expected in the oliguric phase, where kidneys fail to filter adequately.

Choice C reason: Tachycardia may occur in AKI due to fluid overload causing increased cardiac workload or electrolyte imbalances like hyperkalemia affecting heart rhythm. However, it is a secondary symptom and less specific than hyperkalemia, which directly results from impaired renal excretion and poses a more immediate risk to cardiac function.

Choice D reason: Tenting of the skin indicates dehydration, which may precede AKI but is not typical in the oliguric phase, where fluid retention is more common due to reduced urine output. Fluid overload leads to edema, not dehydration, making skin tenting an unlikely finding in this phase of AKI.

Choice A reason: Hypoglycemia involves low blood glucose, causing symptoms like shakiness or confusion, not muscle weakness or arrhythmias. Hypertonic glucose and insulin would worsen hypoglycemia by increasing glucose uptake, and sodium bicarbonate is irrelevant. These symptoms and treatments align with hyperkalemia, not low glucose levels, in renal failure.

Choice B reason: Hyperkalemia, common in acute renal failure due to impaired potassium excretion, causes muscle weakness and cardiac arrhythmias by altering membrane potentials. Hypertonic glucose and insulin drive potassium into cells, while sodium bicarbonate corrects acidosis, stabilizing cardiac membranes, making this the targeted complication for the prescribed treatment.

Choice C reason: Hypernatremia (high sodium) causes neurological symptoms like confusion, not muscle weakness or arrhythmias. The prescribed treatments do not address sodium levels; insulin and glucose manage potassium, and bicarbonate corrects acidosis. Hypernatremia is not a primary concern in acute renal failure with these symptoms.

Choice D reason: Hypokalemia (low potassium) causes muscle weakness and arrhythmias but is rare in acute renal failure, where hyperkalemia is typical due to reduced excretion. The prescribed treatments aim to lower potassium, not increase it, making hypokalemia an incorrect target for this therapy in the context of renal failure.