A 15 kg child has a urine output of 360 mL during the past 12 hours.

Calculate the child’s urine output in mL/kg/hour for this period of time (Do not enter units).

Choice A: Increase Sodium Intake

Increasing sodium intake is generally not recommended for patients with renal disorders. High sodium intake can lead to fluid retention, hypertension, and worsening of kidney function. Patients with renal disorders are often advised to follow a low-sodium diet to manage these risks.

Choice B: Monitor Intake/Output

Monitoring intake and output (I/O) is crucial for patients with renal disorders. It helps in assessing kidney function and fluid balance. Accurate measurement of fluid intake and urine output can provide valuable information about the patient’s hydration status and kidney function. This intervention is essential for preventing fluid overload and dehydration.

Choice C: Daily Weights

Daily weights are an important intervention for patients with renal disorders. Monitoring weight daily helps in detecting fluid retention early. Sudden weight gain can indicate fluid overload, which is a common issue in patients with kidney problems. By tracking daily weights, healthcare providers can make timely adjustments to the patient’s treatment plan.

Choice D: Monitor Blood Pressure

Monitoring blood pressure is a key intervention for patients with renal disorders. Hypertension is both a cause and a consequence of kidney disease. Regular monitoring of blood pressure helps in managing hypertension and preventing further damage to the kidneys. It also aids in assessing the effectiveness of antihypertensive medications.

Choice A reason:

Extracellular hyperglycemia refers to high blood sugar levels outside the cells. While hyperglycemia is a common feature of diabetic ketoacidosis (DKA), it is not the direct cause of the condition. DKA occurs when the body cannot use glucose for energy due to a lack of insulin, leading to the breakdown of fat and protein for energy, which produces ketones and causes acidosis.

Choice B Reason:

Hypoglycemia, or low blood sugar, is not associated with diabetic ketoacidosis. In fact, DKA is characterized by high blood sugar levels. Hypoglycemia is a separate condition that can occur in individuals with diabetes, particularly if they take too much insulin or skip meals.

Choice C Reason:

The use of fat and protein as an energy source is the primary mechanism behind diabetic ketoacidosis. When insulin is deficient, the body cannot utilize glucose for energy. As a result, it breaks down fat and protein to produce energy. This process generates ketones, which are acidic and lead to the development of ketoacidosis.

Choice D Reason:

Increased cellular metabolism of glucose is not related to diabetic ketoacidosis. In DKA, the problem is that cells cannot use glucose effectively due to a lack of insulin. Instead, the body resorts to breaking down fat and protein for energy, leading to the production of ketones and acidosis.